K. Murali Krishna Rao
Health Effects Laboratory Division
National Institute for Occupational Safety and Health
Morgantown
West Virginia
USA
Name/email consistency: medium
- Exposure in vivo to silica or lipopolysaccharide produces transient or sustained upregulation, respectively, of PYPAF7 and MEFV genes in bronchoalveolar lavage cells in rats. Rao, K.M., Meighan, T. J. Toxicol. Environ. Health Part A (2006)
- Time course of gene expression of inflammatory mediators in rat lung after diesel exhaust particle exposure. Rao, K.M., Ma, J.Y., Meighan, T., Barger, M.W., Pack, D., Vallyathan, V. Environ. Health Perspect. (2005)
- The sources of inflammatory mediators in the lung after silica exposure. Rao, K.M., Porter, D.W., Meighan, T., Castranova, V. Environ. Health Perspect. (2004)
- Role of mitogen-activated protein kinase activation in the production of inflammatory mediators: differences between primary rat alveolar macrophages and macrophage cell lines. Rao, K.M., Meighan, T., Bowman, L. J. Toxicol. Environ. Health Part A (2002)
- MAP kinase activation in macrophages. Rao, K.M. J. Leukoc. Biol. (2001)
- Molecular mechanisms regulating iNOS expression in various cell types. Rao, K.M. J. Toxicol. Environ. Health. B. Crit. Rev (2000)