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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Masuko Katoh

Genetics and Cell Biology Section

National Cancer Center

5-1-1 Tsukiji

Chuo-ku

Japan

[email]@umin.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Genetics and Cell Biology Section, National Cancer Center, 5-1-1 Tsukiji, Chuo-ku, Japan. 2007 - 2011
  • M&M Medical BioInformatics, Hongo 113-0033, Japan. 2007 - 2009

References

  1. Network of WNT and other regulatory signaling cascades in pluripotent stem cells and cancer stem cells. Katoh, M. Curr. Pharm. Biotechnol (2011) [Pubmed]
  2. Genetic alterations of FGF receptors: an emerging field in clinical cancer diagnostics and therapeutics. Katoh, M. Expert. Rev. Anticancer. Ther (2010) [Pubmed]
  3. FGFR2 abnormalities underlie a spectrum of bone, skin, and cancer pathologies. Katoh, M. J. Invest. Dermatol. (2009) [Pubmed]
  4. Transcriptional mechanisms of WNT5A based on NF-kappaB, Hedgehog, TGFbeta, and Notch signaling cascades. Katoh, M., Katoh, M. Int. J. Mol. Med. (2009) [Pubmed]
  5. Transcriptional regulation of WNT2B based on the balance of Hedgehog, Notch, BMP and WNT signals. Katoh, M., Katoh, M. Int. J. Oncol. (2009) [Pubmed]
  6. Integrative genomic analyses on GLI2: mechanism of Hedgehog priming through basal GLI2 expression, and interaction map of stem cell signaling network with P53. Katoh, Y., Katoh, M. Int. J. Oncol. (2008) [Pubmed]
  7. Cancer genomics and genetics of FGFR2 (Review). Katoh, M. Int. J. Oncol. (2008) [Pubmed]
  8. RNA technology targeted to the WNT signaling pathway. Katoh, M. Cancer Biol. Ther. (2008) [Pubmed]
  9. WNT signaling in stem cell biology and regenerative medicine. Katoh, M. Curr. Drug. Targets (2008) [Pubmed]
  10. Networking of WNT, FGF, Notch, BMP, and Hedgehog signaling pathways during carcinogenesis. Katoh, M. Stem. Cell. Rev (2007) [Pubmed]
  11. Comparative integromics on non-canonical WNT or planar cell polarity signaling molecules: transcriptional mechanism of PTK7 in colorectal cancer and that of SEMA6A in undifferentiated ES cells. Katoh, M., Katoh, M. Int. J. Mol. Med. (2007) [Pubmed]
  12. Integrative genomic analyses on HES/HEY family: Notch-independent HES1, HES3 transcription in undifferentiated ES cells, and Notch-dependent HES1, HES5, HEY1, HEY2, HEYL transcription in fetal tissues, adult tissues, or cancer. Katoh, M., Katoh, M. Int. J. Oncol. (2007) [Pubmed]
  13. Dysregulation of stem cell signaling network due to germline mutation, SNP, Helicobacter pylori infection, epigenetic change and genetic alteration in gastric cancer. Katoh, M. Cancer Biol. Ther. (2007) [Pubmed]
  14. STAT3-induced WNT5A signaling loop in embryonic stem cells, adult normal tissues, chronic persistent inflammation, rheumatoid arthritis and cancer (Review). Katoh, M., Katoh, M. Int. J. Mol. Med. (2007) [Pubmed]
  15. Conserved POU-binding site linked to SP1-binding site within FZD5 promoter: Transcriptional mechanisms of FZD5 in undifferentiated human ES cells, fetal liver/spleen, adult colon, pancreatic islet, and diffuse-type gastric cancer. Katoh, Y., Katoh, M. Int. J. Oncol. (2007) [Pubmed]
  16. Comparative integromics on FZD7 orthologs: conserved binding sites for PU.1, SP1, CCAAT-box and TCF/LEF/SOX transcription factors within 5'-promoter region of mammalian FZD7 orthologs. Katoh, M., Katoh, M. Int. J. Mol. Med. (2007) [Pubmed]
 
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