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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Sumathy Mohan

Department of Pathology

University of Texas Health Science Center at San Antonio

7703 Floyd Curl Dr.

San Antonio

USA

[email]@uthscsa.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Pathology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, USA. 2003 - 2009

References

  1. High glucose-induced IKK-Hsp-90 interaction contributes to endothelial dysfunction. Mohan, S., Konopinski, R., Yan, B., Centonze, V.E., Natarajan, M. Am. J. Physiol., Cell Physiol. (2009) [Pubmed]
  2. Diabetic eNOS knockout mice develop distinct macro- and microvascular complications. Mohan, S., Reddick, R.L., Musi, N., Horn, D.A., Yan, B., Prihoda, T.J., Natarajan, M., Abboud-Werner, S.L. Lab. Invest. (2008) [Pubmed]
  3. Low shear stress preferentially enhances IKK activity through selective sources of ROS for persistent activation of NF-kappaB in endothelial cells. Mohan, S., Koyoma, K., Thangasamy, A., Nakano, H., Glickman, R.D., Mohan, N. Am. J. Physiol., Cell Physiol. (2007) [Pubmed]
  4. IkappaBalpha-dependent regulation of low-shear flow-induced NF-kappa B activity: role of nitric oxide. Mohan, S., Hamuro, M., Sorescu, G.P., Koyoma, K., Sprague, E.A., Jo, H., Valente, A.J., Prihoda, T.J., Natarajan, M. Am. J. Physiol., Cell Physiol. (2003) [Pubmed]
  5. High glucose induced NF-kappaB DNA-binding activity in HAEC is maintained under low shear stress but inhibited under high shear stress: role of nitric oxide. Mohan, S., Hamuro, M., Koyoma, K., Sorescu, G.P., Jo, H., Natarajan, M. Atherosclerosis (2003) [Pubmed]
 
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