Sumathy Mohan
Department of Pathology
University of Texas Health Science Center at San Antonio
7703 Floyd Curl Dr.
San Antonio
USA
Name/email consistency: high
- High glucose-induced IKK-Hsp-90 interaction contributes to endothelial dysfunction. Mohan, S., Konopinski, R., Yan, B., Centonze, V.E., Natarajan, M. Am. J. Physiol., Cell Physiol. (2009)
- Diabetic eNOS knockout mice develop distinct macro- and microvascular complications. Mohan, S., Reddick, R.L., Musi, N., Horn, D.A., Yan, B., Prihoda, T.J., Natarajan, M., Abboud-Werner, S.L. Lab. Invest. (2008)
- Low shear stress preferentially enhances IKK activity through selective sources of ROS for persistent activation of NF-kappaB in endothelial cells. Mohan, S., Koyoma, K., Thangasamy, A., Nakano, H., Glickman, R.D., Mohan, N. Am. J. Physiol., Cell Physiol. (2007)
- IkappaBalpha-dependent regulation of low-shear flow-induced NF-kappa B activity: role of nitric oxide. Mohan, S., Hamuro, M., Sorescu, G.P., Koyoma, K., Sprague, E.A., Jo, H., Valente, A.J., Prihoda, T.J., Natarajan, M. Am. J. Physiol., Cell Physiol. (2003)
- High glucose induced NF-kappaB DNA-binding activity in HAEC is maintained under low shear stress but inhibited under high shear stress: role of nitric oxide. Mohan, S., Hamuro, M., Koyoma, K., Sorescu, G.P., Jo, H., Natarajan, M. Atherosclerosis (2003)