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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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Martin Poot

Department of Pathology

University of Washington

Seattle 98195-7705

USA

[email]@u.washington.edu

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Affiliation

Department of Pathology, University of Washington, Seattle 98195-7705, USA. 1999 - 2004

References

Distinct functions for WRN and TP53 in a shared pathway of cellular response to 1-beta-D-arabinofuranosylcytosine and bleomycin. Poot, M., Jin, X., Hill, J.P., Gollahon, K.A., Rabinovitch, P.S. Exp. Cell Res. (2004) [Pubmed]
Werner syndrome diploid fibroblasts are sensitive to 4-nitroquinoline-N-oxide and 8-methoxypsoralen: implications for the disease phenotype. Poot, M., Gollahon, K.A., Emond, M.J., Silber, J.R., Rabinovitch, P.S. FASEB J. (2002) [Pubmed]
Werner syndrome cells are sensitive to DNA cross-linking drugs. Poot, M., Yom, J.S., Whang, S.H., Kato, J.T., Gollahon, K.A., Rabinovitch, P.S. FASEB J. (2001) [Pubmed]
Werner syndrome lymphoblastoid cells are sensitive to camptothecin-induced apoptosis in S-phase. Poot, M., Gollahon, K.A., Rabinovitch, P.S. Hum. Genet. (1999) [Pubmed]
Detection of changes in mitochondrial function during apoptosis by simultaneous staining with multiple fluorescent dyes and correlated multiparameter flow cytometry. Poot, M., Pierce, R.H. Cytometry (1999) [Pubmed]