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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

M.S. Sheikh

Division of Basic Sciences

NCI, National Institutes of Health

Bethesda MD 20892



Name/email consistency: high



  • Division of Basic Sciences, NCI, National Institutes of Health, Bethesda MD 20892, USA. 1999
  • Laboratory of Molecular Pharmacology, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA. 1996 - 1998


  1. Cloning and characterization of a human genotoxic and endoplasmic reticulum stress-inducible cDNA that encodes translation initiation factor 1(eIF1(A121/SUI1)). Sheikh, M.S., Fernandez-Salas, E., Yu, M., Hussain, A., Dinman, J.D., Peltz, S.W., Huang, Y., Fornace, A.J. J. Biol. Chem. (1999) [Pubmed]
  2. p53-dependent and -independent regulation of the death receptor KILLER/DR5 gene expression in response to genotoxic stress and tumor necrosis factor alpha. Sheikh, M.S., Burns, T.F., Huang, Y., Wu, G.S., Amundson, S., Brooks, K.S., Fornace, A.J., el-Deiry, W.S. Cancer Res. (1998) [Pubmed]
  3. Identification of several human homologs of hamster DNA damage-inducible transcripts. Cloning and characterization of a novel UV-inducible cDNA that codes for a putative RNA-binding protein. Sheikh, M.S., Carrier, F., Papathanasiou, M.A., Hollander, M.C., Zhan, Q., Yu, K., Fornace, A.J. J. Biol. Chem. (1997) [Pubmed]
  4. Cell cycle-independent regulation of p21Waf1/Cip1 and retinoblastoma protein during okadaic acid-induced apoptosis is coupled with induction of Bax protein in human breast carcinoma cells. Sheikh, M.S., Garcia, M., Zhan, Q., Liu, Y., Fornace, A.J. Cell Growth Differ. (1996) [Pubmed]
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