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Naim Akhtar Khan

University of Burgundy

Department of Physiology

UPRES EA4183 Lipids and Cell Signalling

Faculty of Life Sciences

France

[email]@u-bourgogne.fr

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • University of Burgundy, Department of Physiology, UPRES EA4183 Lipids and Cell Signalling, Faculty of Life Sciences, France. 2010
  • Faculté des Sciences de la Vie, Université de Bourgogne, Dijon 21000, France. 2009
  • Department of Physiology, UPRES Lipides & Signalisation Cellulaire, Université de Bourgogne, Dijon, France. 1999 - 2007
  • UPRES EA 4183 Lipides & Signalisation Cellulaire, Université de Bourgogne, Faculté des Sciences de la vie, 6, Boulevard Gabriel, France. 1999 - 2007
  • Unité Propre de Recherche de l'Enseignement Supérieure-Lipides & Nutrition, Faculty of Life Sciences, University of Burgundy, Dijon, France. 2006

References

  1. Polyunsaturated fatty acids in the modulation of T-cell signalling. Akhtar Khan, N. Prostaglandins Leukot. Essent. Fatty Acids (2010) [Pubmed]
  2. Oro-sensory perception of dietary lipids: new insights into the fat taste transduction. Khan, N.A., Besnard, P. Biochim. Biophys. Acta (2009) [Pubmed]
  3. Role of lipids and fatty acids in macrosomic offspring of diabetic pregnancy. Khan, N.A. Cell Biochem. Biophys. (2007) [Pubmed]
  4. Role of T-cells in diabetic pregnancy and macrosomia. Khan, N.A. Indian J. Biochem. Biophys. (2007) [Pubmed]
  5. N-3 fatty acids modulate Th1 and Th2 dichotomy in diabetic pregnancy and macrosomia. Khan, N.A., Yessoufou, A., Kim, M., Hichami, A. J. Autoimmun. (2006) [Pubmed]
  6. Docosahexaenoic acid inhibits cancer cell growth via p27Kip1, CDK2, ERK1/ERK2, and retinoblastoma phosphorylation. Khan, N.A., Nishimura, K., Aires, V., Yamashita, T., Oaxaca-Castillo, D., Kashiwagi, K., Igarashi, K. J. Lipid Res. (2006) [Pubmed]
  7. 5-HT3 receptor-channels coupled with Na+ influx in human T cells: role in T cell activation. Khan, N.A., Poisson, J.P. J. Neuroimmunol. (1999) [Pubmed]
  8. Ionotrophic 5-hydroxytryptamine type 3 receptor activates the protein kinase C-dependent phospholipase D pathway in human T-cells. Khan, N.A., Hichami, A. Biochem. J. (1999) [Pubmed]
 
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