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Takahiko Nakagawa

Division of Nephrology

Hypertension and Transplantation

University of Florida

PO Box 100224

USA

[email]@medicine.ufl.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Nephrology, Hypertension and Transplantation, University of Florida, PO Box 100224, USA. 2004 - 2008
  • Division of Nephrology-Medicine, Baylor College of Medicine, Houston, Texas, USA. 2004

References

  1. The conundrum of hyperuricemia, metabolic syndrome, and renal disease. Nakagawa, T., Cirillo, P., Sato, W., Gersch, M., Sautin, Y., Roncal, C., Mu, W., Sánchez-Lozada, L.G., Johnson, R.J. Intern. Emerg. Med (2008) [Pubmed]
  2. Uncoupling of VEGF with NO as a mechanism for diabetic nephropathy. Nakagawa, T. Diabetes Res. Clin. Pract. (2008) [Pubmed]
  3. Diabetic endothelial nitric oxide synthase knockout mice develop advanced diabetic nephropathy. Nakagawa, T., Sato, W., Glushakova, O., Heinig, M., Clarke, T., Campbell-Thompson, M., Yuzawa, Y., Atkinson, M.A., Johnson, R.J., Croker, B. J. Am. Soc. Nephrol. (2007) [Pubmed]
  4. Uncoupling of the VEGF-endothelial nitric oxide axis in diabetic nephropathy: an explanation for the paradoxical effects of VEGF in renal disease. Nakagawa, T. Am. J. Physiol. Renal Physiol. (2007) [Pubmed]
  5. A breakthrough in diabetic nephropathy: the role of endothelial dysfunction. Nakagawa, T., Segal, M., Croker, B., Johnson, R.J. Nephrol. Dial. Transplant. (2007) [Pubmed]
  6. A causal role for uric acid in fructose-induced metabolic syndrome. Nakagawa, T., Hu, H., Zharikov, S., Tuttle, K.R., Short, R.A., Glushakova, O., Ouyang, X., Feig, D.I., Block, E.R., Herrera-Acosta, J., Patel, J.M., Johnson, R.J. Am. J. Physiol. Renal Physiol. (2006) [Pubmed]
  7. Uric acid--a uremic toxin?. Nakagawa, T., Mazzali, M., Kang, D.H., Sánchez-Lozada, L.G., Herrera-Acosta, J., Johnson, R.J. Blood Purif. (2006) [Pubmed]
  8. Uncoupling of vascular endothelial growth factor with nitric oxide as a mechanism for diabetic vasculopathy. Nakagawa, T., Sato, W., Sautin, Y.Y., Glushakova, O., Croker, B., Atkinson, M.A., Tisher, C.C., Johnson, R.J. J. Am. Soc. Nephrol. (2006) [Pubmed]
  9. Unearthing uric acid: an ancient factor with recently found significance in renal and cardiovascular disease. Nakagawa, T., Kang, D.H., Feig, D., Sanchez-Lozada, L.G., Srinivas, T.R., Sautin, Y., Ejaz, A.A., Segal, M., Johnson, R.J. Kidney Int. (2006) [Pubmed]
  10. Role of ERK1/2 and p38 mitogen-activated protein kinases in the regulation of thrombospondin-1 by TGF-beta1 in rat proximal tubular cells and mouse fibroblasts. Nakagawa, T., Lan, H.Y., Glushakova, O., Zhu, H.J., Kang, D.H., Schreiner, G.F., Böttinger, E.P., Johnson, R.J., Sautin, Y.Y. J. Am. Soc. Nephrol. (2005) [Pubmed]
  11. Hypothesis: fructose-induced hyperuricemia as a causal mechanism for the epidemic of the metabolic syndrome. Nakagawa, T., Tuttle, K.R., Short, R.A., Johnson, R.J. Nature Clinical Practice. Nephrology (2005) [Pubmed]
  12. Differential regulation of VEGF by TGF-beta and hypoxia in rat proximal tubular cells. Nakagawa, T., Lan, H.Y., Zhu, H.J., Kang, D.H., Schreiner, G.F., Johnson, R.J. Am. J. Physiol. Renal Physiol. (2004) [Pubmed]
  13. TGF-beta induces proangiogenic and antiangiogenic factors via parallel but distinct Smad pathways. Nakagawa, T., Li, J.H., Garcia, G., Mu, W., Piek, E., Böttinger, E.P., Chen, Y., Zhu, H.J., Kang, D.H., Schreiner, G.F., Lan, H.Y., Johnson, R.J. Kidney Int. (2004) [Pubmed]
 
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