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Retha R. Newbold

Developmental Endocrinology and Endocrine Disruptor Section

Laboratory of Molecular Toxicology

National Institute of Environmental Health Sciences

NIH, DHHS, Research Triangle Park

United States

[email]@niehs.nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Developmental Endocrinology and Endocrine Disruptor Section, Laboratory of Molecular Toxicology, National Institute of Environmental Health Sciences, NIH, DHHS, Research Triangle Park, United States. 1997 - 2009
  • Laboratory of Molecular Toxicology, National Institute of Environmental Health Sciences, National Institutes of Health, Department of Health and Human Services, USA. 2009
  • Developmental Endocrinology Section, Laboratory of Toxicology, Environmental Toxicology Program, Division of Intramural Research, USA. 2001 - 2004

References

  1. Environmental estrogens and obesity. Newbold, R.R., Padilla-Banks, E., Jefferson, W.N. Mol. Cell. Endocrinol. (2009) [Pubmed]
  2. Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life. Newbold, R.R., Jefferson, W.N., Padilla-Banks, E. Environ. Health Perspect. (2009) [Pubmed]
  3. Effects of endocrine disruptors on obesity. Newbold, R.R., Padilla-Banks, E., Jefferson, W.N., Heindel, J.J. Int. J. Androl. (2008) [Pubmed]
  4. Prenatal exposure to diethylstilbestrol (DES). Newbold, R.R. Fertil. Steril. (2008) [Pubmed]
  5. Developmental exposure to endocrine disruptors and the obesity epidemic. Newbold, R.R., Padilla-Banks, E., Snyder, R.J., Phillips, T.M., Jefferson, W.N. Reprod. Toxicol. (2007) [Pubmed]
  6. Perinatal exposure to environmental estrogens and the development of obesity. Newbold, R.R., Padilla-Banks, E., Snyder, R.J., Jefferson, W.N. Mol. Nutr. Food. Res (2007) [Pubmed]
  7. Long-term adverse effects of neonatal exposure to bisphenol A on the murine female reproductive tract. Newbold, R.R., Jefferson, W.N., Padilla-Banks, E. Reprod. Toxicol. (2007) [Pubmed]
  8. Adverse effects of the model environmental estrogen diethylstilbestrol are transmitted to subsequent generations. Newbold, R.R., Padilla-Banks, E., Jefferson, W.N. Endocrinology (2006) [Pubmed]
  9. Developmental exposure to diethylstilbestrol (DES) alters uterine response to estrogens in prepubescent mice: low versus high dose effects. Newbold, R.R., Jefferson, W.N., Padilla-Banks, E., Haseman, J. Reprod. Toxicol. (2004) [Pubmed]
  10. Lessons learned from perinatal exposure to diethylstilbestrol. Newbold, R.R. Toxicol. Appl. Pharmacol. (2004) [Pubmed]
  11. Uterine adenocarcinoma in mice treated neonatally with genistein. Newbold, R.R., Banks, E.P., Bullock, B., Jefferson, W.N. Cancer Res. (2001) [Pubmed]
  12. Cell response endpoints enhance sensitivity of the immature mouse uterotropic assay. Newbold, R.R., Jefferson, W.N., Padilla-Banks, E., Walker, V.R., Pena, D.S. Reprod. Toxicol. (2001) [Pubmed]
  13. Induction of uterine adenocarcinoma in CD-1 mice by catechol estrogens. Newbold, R.R., Liehr, J.G. Cancer Res. (2000) [Pubmed]
  14. Proliferative lesions and reproductive tract tumors in male descendants of mice exposed developmentally to diethylstilbestrol. Newbold, R.R., Hanson, R.B., Jefferson, W.N., Bullock, B.C., Haseman, J., McLachlan, J.A. Carcinogenesis (2000) [Pubmed]
  15. Increased tumors but uncompromised fertility in the female descendants of mice exposed developmentally to diethylstilbestrol. Newbold, R.R., Hanson, R.B., Jefferson, W.N., Bullock, B.C., Haseman, J., McLachlan, J.A. Carcinogenesis (1998) [Pubmed]
  16. Ontogeny of lactoferrin in the developing mouse uterus: a marker of early hormone response. Newbold, R.R., Hanson, R.B., Jefferson, W.N. Biol. Reprod. (1997) [Pubmed]
 
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