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Norberto C. Gonzalez

Dept. of Molecular and Integrative Physiology

Mail Stop 3043

Univ. of Kansas Medical Center

3901 Rainbow Blvd

[email]@kumc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Dept. of Molecular and Integrative Physiology, Mail Stop 3043, Univ. of Kansas Medical Center, 3901 Rainbow Blvd. 2006 - 2011

References

  1. Renin released from mast cells activated by circulating MCP-1 initiates the microvascular phase of the systemic inflammation of alveolar hypoxia. Chao, J., Blanco, G., Wood, J.G., Gonzalez, N.C. Am. J. Physiol. Heart Circ. Physiol. (2011) [Pubmed]
  2. Monocyte chemoattractant protein-1 released from alveolar macrophages mediates the systemic inflammation of acute alveolar hypoxia. Chao, J., Donham, P., van Rooijen, N., Wood, J.G., Gonzalez, N.C. Am. J. Respir. Cell Mol. Biol. (2011) [Pubmed]
  3. Alveolar hypoxia-induced systemic inflammation: what low PO(2) does and does not do. Gonzalez, N.C., Wood, J.G. Adv. Exp. Med. Biol. (2010) [Pubmed]
  4. Alveolar macrophages are necessary for the systemic inflammation of acute alveolar hypoxia. Gonzalez, N.C., Allen, J., Blanco, V.G., Schmidt, E.J., van Rooijen, N., Wood, J.G. J. Appl. Physiol. (2007) [Pubmed]
  5. Role of the renin-angiotensin system in the systemic microvascular inflammation of alveolar hypoxia. Gonzalez, N.C., Allen, J., Schmidt, E.J., Casillan, A.J., Orth, T., Wood, J.G. Am. J. Physiol. Heart Circ. Physiol. (2007) [Pubmed]
  6. Systemic oxygen transport in rats artificially selected for running endurance. Gonzalez, N.C., Howlett, R.A., Henderson, K.K., Koch, L.G., Britton, S.L., Wagner, H.E., Favret, F., Wagner, P.D. Respir. Physiol. Neurobiol (2006) [Pubmed]
  7. Continued divergence in VO2max of rats artificially selected for running endurance is mediated by greater convective blood O2 delivery. Gonzalez, N.C., Kirkton, S.D., Howlett, R.A., Britton, S.L., Koch, L.G., Wagner, H.E., Wagner, P.D. J. Appl. Physiol. (2006) [Pubmed]
 
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