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Nima Sharifi

Medical Oncology Branch

National Cancer Institute

Bethesda

Maryland 20892

USA

[email]@nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Medical Oncology Branch, National Cancer Institute, Bethesda, Maryland 20892, USA. 2007 - 2008
  • Cancer Stem Cell Section, Laboratory of Cancer Prevention, National Cancer Institute at Frederick, Center for Cancer Research, USA. 2007
  • Cytokine Molecular Mechanisms Section, Laboratory of Molecular Immunoregulation, Center for Cancer Research, National Cancer Institute at Frederick, USA. 2006

References

  1. Secondary hormonal therapy for prostate cancer: what lies on the horizon?. Sharifi, N., Dahut, W.L., Figg, W.D. BJU Int. (2008) [Pubmed]
  2. Transforming growth factor-beta receptor III downregulation in prostate cancer: is inhibin B a tumor suppressor in prostate? Sharifi, N., Lechleider, R.J., Farrar, W.L. J. Mol. Endocrinol. (2007) [Pubmed]
  3. A bifunctional colchicinoid that binds to the androgen receptor. Sharifi, N., Hamel, E., Lill, M.A., Risbood, P., Kane, C.T., Hossain, M.T., Jones, A., Dalton, J.T., Farrar, W.L. Mol. Cancer Ther. (2007) [Pubmed]
  4. Androgen receptor modulation: lessons learned from beyond the prostate. Sharifi, N., Figg, W.D. Cancer Biol. Ther. (2007) [Pubmed]
  5. TGFBR3 loss and consequences in prostate cancer. Sharifi, N., Hurt, E.M., Kawasaki, B.T., Farrar, W.L. Prostate (2007) [Pubmed]
  6. Perturbations in hypoxia detection: a shared link between hereditary and sporadic tumor formation?. Sharifi, N., Farrar, W.L. Med. Hypotheses (2006) [Pubmed]
  7. Androgen receptor as a therapeutic target for androgen independent prostate cancer. Sharifi, N., Farrar, W.L. Am. J. Ther (2006) [Pubmed]
 
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