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Jerzy-Roch Nofer

Jerzy-Roch Nofer

MD, MBA, Centrum für Laboratoriumsmedizin

Universitätsklinikum Münster

E-mail:

[email]@uni-muenster.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Jerzy-Roch Nofer, MD, MBA, Centrum für Laboratoriumsmedizin, Universitätsklinikum Münster, E-mail:. 2011 - 2012
  • Center for Laboratory Medicine, University Hospital Münster, Münster, Germany. 2010
  • Institut für Klinische Chemie und Laboratoriumsmedizin, Zentrallaboratorium, Westfälische Wilhelms-Universität, A. Schweitzer Str 33, Germany. 2000 - 2007
  • Institut für Klinische Chemie und Laboratoriumsmedizin, Westfälische Wilhelms-Universität and the Institut für Arterioskleroseforschung an der Universität Münster, 48129 Münster, Germany. 2001

References

  1. Sphingosine kinase inhibition exerts both pro- and anti-atherogenic effects in low-density lipoprotein receptor-deficient (LDL-R-/-) mice. Poti, F., Bot, M., Costa, S., Bergonzini, V., Maines, L., Varga, G., Freise, H., Robenek, H., Simoni, M., Nofer, J.R. Thromb. Haemost. (2012) [Pubmed]
  2. Apolipoprotein e induces antiinflammatory phenotype in macrophages. Baitsch, D., Bock, H.H., Engel, T., Telgmann, R., Müller-Tidow, C., Varga, G., Bot, M., Herz, J., Robenek, H., von Eckardstein, A., Nofer, J.R. Arterioscler. Thromb. Vasc. Biol. (2011) [Pubmed]
  3. High-density lipoproteins, platelets and the pathogenesis of atherosclerosis. Nofer, J.R., Brodde, M.F., Kehrel, B.E. Clin. Exp. Pharmacol. Physiol. (2010) [Pubmed]
  4. FTY720, a synthetic sphingosine 1 phosphate analogue, inhibits development of atherosclerosis in low-density lipoprotein receptor-deficient mice. Nofer, J.R., Bot, M., Brodde, M., Taylor, P.J., Salm, P., Brinkmann, V., van Berkel, T., Assmann, G., Biessen, E.A. Circulation (2007) [Pubmed]
  5. Low density lipoproteins inhibit the Na+/H+ antiport in human platelets via activation of p38MAP kinase. Nofer, J.R., Noll, C., Feuerborn, R., Assmann, G., Tepel, M. Biochem. Biophys. Res. Commun. (2006) [Pubmed]
  6. Apolipoprotein A-I activates Cdc42 signaling through the ABCA1 transporter. Nofer, J.R., Remaley, A.T., Feuerborn, R., Wolinnska, I., Engel, T., von Eckardstein, A., Assmann, G. J. Lipid Res. (2006) [Pubmed]
  7. Atheroprotective effects of high-density lipoprotein-associated lysosphingolipids. Nofer, J.R., Assmann, G. Trends Cardiovasc. Med. (2005) [Pubmed]
  8. Current understanding of the role of high-density lipoproteins in atherosclerosis and senescence. Nofer, J.R., Walter, M., Assmann, G. Expert. Rev. Cardiovasc. Ther (2005) [Pubmed]
  9. Impaired platelet activation in familial high density lipoprotein deficiency (Tangier disease). Nofer, J.R., Herminghaus, G., Brodde, M., Morgenstern, E., Rust, S., Engel, T., Seedorf, U., Assmann, G., Bluethmann, H., Kehrel, B.E. J. Biol. Chem. (2004) [Pubmed]
  10. High density lipoprotein-associated lysosphingolipids reduce E-selectin expression in human endothelial cells. Nofer, J.R., Geigenmüller, S., Göpfert, C., Assmann, G., Buddecke, E., Schmidt, A. Biochem. Biophys. Res. Commun. (2003) [Pubmed]
  11. Involvement of Cdc42 signaling in apoA-I-induced cholesterol efflux. Nofer, J.R., Feuerborn, R., Levkau, B., Sokoll, A., Seedorf, U., Assmann, G. J. Biol. Chem. (2003) [Pubmed]
  12. HDL and arteriosclerosis: beyond reverse cholesterol transport. Nofer, J.R., Kehrel, B., Fobker, M., Levkau, B., Assmann, G., von Eckardstein, A. Atherosclerosis (2002) [Pubmed]
  13. High density lipoproteins induce cell cycle entry in vascular smooth muscle cells via mitogen activated protein kinase-dependent pathway. Nofer, J.R., Junker, R., Pulawski, E., Fobker, M., Levkau, B., von Eckardstein, A., Seedorf, U., Assmann, G., Walter, M. Thromb. Haemost. (2001) [Pubmed]
  14. Suppression of endothelial cell apoptosis by high density lipoproteins (HDL) and HDL-associated lysosphingolipids. Nofer, J.R., Levkau, B., Wolinska, I., Junker, R., Fobker, M., von Eckardstein, A., Seedorf, U., Assmann, G. J. Biol. Chem. (2001) [Pubmed]
  15. D609-phosphatidylcholine-specific phospholipase C inhibitor attenuates thapsigargin-induced sodium influx in human lymphocytes. Nofer, J.R., Junker, R., Seedorf, U., Assmann, G., Zidek, W., Tepel, M. Cell. Signal. (2000) [Pubmed]
 
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