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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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Olof H. Hultgren

Department of Clinical Immunology

Sahlgren's University Hospital

Göteborg

Sweden

[email]@immuno.gu.se

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Affiliations

Department of Clinical Immunology, Sahlgren's University Hospital, Göteborg, Sweden. 2004 - 2007
Department of Rheumatology, University of Göteborg, Sweden. 1998 - 2002

References

Serum concentration of interleukin-18 is up-regulated in patients with ANCA-associated vasculitis. Hultgren, O., Andersson, B., Hahn-Zoric, M., Almroth, G. Autoimmunity (2007) [Pubmed]
Serum interleukin-1 receptor antagonist is an early indicator of colitis onset in Galphai2-deficient mice. Hultgren, O.H., Berglund, M., Bjursten, M., Hultgren Hörnquist, E. World J. Gastroenterol. (2006) [Pubmed]
T-box transcription-factor-deficient mice display increased joint pathology and failure of infection control during staphylococcal arthritis. Hultgren, O.H., Verdrengh, M., Tarkowski, A. Microbes Infect. (2004) [Pubmed]
Critical role of signaling through IL-1 receptor for development of arthritis and sepsis during Staphylococcus aureus infection. Hultgren, O.H., Svensson, L., Tarkowski, A. J. Immunol. (2002) [Pubmed]
Role of IL-12 in Staphylococcus aureus-triggered arthritis and sepsis. Hultgren, O.H., Stenson, M., Tarkowski, A. Arthritis Res. (2001) [Pubmed]
Leptin in septic arthritis: decreased levels during infection and amelioration of disease activity upon its administration. Hultgren, O.H., Tarkowski, A. Arthritis Res. (2001) [Pubmed]
Outcome of Staphylococcus aureus-triggered sepsis and arthritis in IL-4-deficient mice depends on the genetic background of the host. Hultgren, O., Kopf, M., Tarkowski, A. Eur. J. Immunol. (1999) [Pubmed]
Staphylococcus aureus-induced septic arthritis and septic death is decreased in IL-4-deficient mice: role of IL-4 as promoter for bacterial growth. Hultgren, O., Kopf, M., Tarkowski, A. J. Immunol. (1998) [Pubmed]
TNF/lymphotoxin-alpha double-mutant mice resist septic arthritis but display increased mortality in response to Staphylococcus aureus. Hultgren, O., Eugster, H.P., Sedgwick, J.D., Körner, H., Tarkowski, A. J. Immunol. (1998) [Pubmed]