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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Pamela L. Rice

Veterans Affairs Medical Center; Denver

USA

[email]@uchsc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Veterans Affairs Medical Center; Denver, USA. 2006
  • Veterans Administration Medical Center, Department of Medicine, University of Colorado Health Sciences Center, and University of Colorado Comprehensive Cancer Center, USA. 2004
  • Department of Medicine, University of Colorado Health Sciences Center, Denver, CO 80262, USA. 2003

References

  1. Sulindac independently modulates extracellular signal-regulated kinase 1/2 and cyclic GMP-dependent protein kinase signaling pathways. Rice, P.L., Peters, S.L., Beard, K.S., Ahnen, D.J. Mol. Cancer Ther. (2006) [Pubmed]
  2. Inhibition of extracellular-signal regulated kinases 1/2 is required for apoptosis of human colon cancer cells in vitro by sulindac metabolites. Rice, P.L., Beard, K.S., Driggers, L.J., Ahnen, D.J. Cancer Res. (2004) [Pubmed]
  3. Sulindac metabolites induce caspase- and proteasome-dependent degradation of beta-catenin protein in human colon cancer cells. Rice, P.L., Kelloff, J., Sullivan, H., Driggers, L.J., Beard, K.S., Kuwada, S., Piazza, G., Ahnen, D.J. Mol. Cancer Ther. (2003) [Pubmed]
 
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