Parri Wentzel
Department of Medical Cell Biology
University of Uppsala
Biomedicum
Sweden
Name/email consistency: high
- Ethanol-induced fetal dysmorphogenesis in the mouse is diminished by high antioxidative capacity of the mother. Wentzel, P., Eriksson, U.J. Toxicol. Sci. (2006)
- Antioxidative treatment diminishes ethanol-induced congenital malformations in the rat. Wentzel, P., Rydberg, U., Eriksson, U.J. Alcohol. Clin. Exp. Res. (2006)
- Folic acid supplementation diminishes diabetes- and glucose-induced dysmorphogenesis in rat embryos in vivo and in vitro. Wentzel, P., Gäreskog, M., Eriksson, U.J. Diabetes (2005)
- A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid. Wentzel, P., Eriksson, U.J. Birth Defects Res. Part A Clin. Mol. Teratol. (2005)
- Maternal diabetes in vivo and high glucose in vitro diminish GAPDH activity in rat embryos. Wentzel, P., Ejdesjö, A., Eriksson, U.J. Diabetes (2003)
- 8-Iso-PGF(2alpha) administration generates dysmorphogenesis and increased lipid peroxidation in rat embryos in vitro. Wentzel, P., Eriksson, U.J. Teratology (2002)
- Developmental damage, increased lipid peroxidation, diminished cyclooxygenase-2 gene expression, and lowered prostaglandin E2 levels in rat embryos exposed to a diabetic environment. Wentzel, P., Welsh, N., Eriksson, U.J. Diabetes (1999)
- Antioxidants diminish developmental damage induced by high glucose and cyclooxygenase inhibitors in rat embryos in vitro. Wentzel, P., Eriksson, U.J. Diabetes (1998)
