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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Parri Wentzel

Department of Medical Cell Biology

University of Uppsala




Name/email consistency: high



  • Department of Medical Cell Biology, University of Uppsala, Biomedicum, Sweden. 1998 - 2006


  1. Ethanol-induced fetal dysmorphogenesis in the mouse is diminished by high antioxidative capacity of the mother. Wentzel, P., Eriksson, U.J. Toxicol. Sci. (2006) [Pubmed]
  2. Antioxidative treatment diminishes ethanol-induced congenital malformations in the rat. Wentzel, P., Rydberg, U., Eriksson, U.J. Alcohol. Clin. Exp. Res. (2006) [Pubmed]
  3. Folic acid supplementation diminishes diabetes- and glucose-induced dysmorphogenesis in rat embryos in vivo and in vitro. Wentzel, P., Gäreskog, M., Eriksson, U.J. Diabetes (2005) [Pubmed]
  4. A diabetes-like environment increases malformation rate and diminishes prostaglandin E(2) in rat embryos: reversal by administration of vitamin E and folic acid. Wentzel, P., Eriksson, U.J. Birth Defects Res. Part A Clin. Mol. Teratol. (2005) [Pubmed]
  5. Maternal diabetes in vivo and high glucose in vitro diminish GAPDH activity in rat embryos. Wentzel, P., Ejdesjö, A., Eriksson, U.J. Diabetes (2003) [Pubmed]
  6. 8-Iso-PGF(2alpha) administration generates dysmorphogenesis and increased lipid peroxidation in rat embryos in vitro. Wentzel, P., Eriksson, U.J. Teratology (2002) [Pubmed]
  7. Developmental damage, increased lipid peroxidation, diminished cyclooxygenase-2 gene expression, and lowered prostaglandin E2 levels in rat embryos exposed to a diabetic environment. Wentzel, P., Welsh, N., Eriksson, U.J. Diabetes (1999) [Pubmed]
  8. Antioxidants diminish developmental damage induced by high glucose and cyclooxygenase inhibitors in rat embryos in vitro. Wentzel, P., Eriksson, U.J. Diabetes (1998) [Pubmed]
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