Patrick Most
Center for Translational Medicine
Department of Medicine
Thomas Jefferson University
Philadelphia
USA
Name/email consistency: high
- S100A1: a novel inotropic regulator of cardiac performance. Transition from molecular physiology to pathophysiological relevance. Most, P., Remppis, A., Pleger, S.T., Katus, H.A., Koch, W.J. Am. J. Physiol. Regul. Integr. Comp. Physiol. (2007)
- Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction. Most, P., Seifert, H., Gao, E., Funakoshi, H., Völkers, M., Heierhorst, J., Remppis, A., Pleger, S.T., DeGeorge, B.R., Eckhart, A.D., Feldman, A.M., Koch, W.J. Circulation (2006)
- Cardiac adenoviral S100A1 gene delivery rescues failing myocardium. Most, P., Pleger, S.T., Völkers, M., Heidt, B., Boerries, M., Weichenhan, D., Löffler, E., Janssen, P.M., Eckhart, A.D., Martini, J., Williams, M.L., Katus, H.A., Remppis, A., Koch, W.J. J. Clin. Invest. (2004)