Patrick Most
Center for Molecular and Translational Cardiology
Heidelberg University Hospital
69120 Heidelberg
Germany
Name/email consistency: high
- S100A1 deficiency impairs postischemic angiogenesis via compromised proangiogenic endothelial cell function and nitric oxide synthase regulation. Most, P., Lerchenmüller, C., Rengo, G., Mahlmann, A., Ritterhoff, J., Rohde, D., Goodman, C., Busch, C.J., Laube, F., Heissenberg, J., Pleger, S.T., Weiss, N., Katus, H.A., Koch, W.J., Peppel, K. Circ. Res. (2013)
- Extracellular S100A1 protein inhibits apoptosis in ventricular cardiomyocytes via activation of the extracellular signal-regulated protein kinase 1/2 (ERK1/2). Most, P., Boerries, M., Eicher, C., Schweda, C., Ehlermann, P., Pleger, S.T., Loeffler, E., Koch, W.J., Katus, H.A., Schoenenberger, C.A., Remppis, A. J. Biol. Chem. (2003)
