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Pavel Krejci

Medical Genetics Institute

Cedars-Sinai Medical Center

Los Angeles

CA 90048

USA

[email]@cshs.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Medical Genetics Institute, Cedars-Sinai Medical Center, Los Angeles, CA 90048, USA. 2007 - 2010
  • Department of Animal Physiology and Immunology, Institute of Experimental Biology, Masaryk University, Brno, Czech Republic. 2008

References

  1. NF449 is a novel inhibitor of fibroblast growth factor receptor 3 (FGFR3) signaling active in chondrocytes and multiple myeloma cells. Krejci, P., Murakami, S., Prochazkova, J., Trantirek, L., Chlebova, K., Ouyang, Z., Aklian, A., Smutny, J., Bryja, V., Kozubik, A., Wilcox, W.R. J. Biol. Chem. (2010) [Pubmed]
  2. Analysis of STAT1 activation by six FGFR3 mutants associated with skeletal dysplasia undermines dominant role of STAT1 in FGFR3 signaling in cartilage. Krejci, P., Salazar, L., Kashiwada, T.A., Chlebova, K., Salasova, A., Thompson, L.M., Bryja, V., Kozubik, A., Wilcox, W.R. PLoS. ONE (2008) [Pubmed]
  3. Simple, mammalian cell-based assay for identification of inhibitors of the Erk MAP kinase pathway. Krejci, P., Pejchalova, K., Wilcox, W.R. Invest. New. Drugs (2007) [Pubmed]
  4. Bisindolylmaleimide I suppresses fibroblast growth factor-mediated activation of Erk MAP kinase in chondrocytes by preventing Shp2 association with the Frs2 and Gab1 adaptor proteins. Krejci, P., Masri, B., Salazar, L., Farrington-Rock, C., Prats, H., Thompson, L.M., Wilcox, W.R. J. Biol. Chem. (2007) [Pubmed]
  5. Fibroblast growth factors 1, 2, 17, and 19 are the predominant FGF ligands expressed in human fetal growth plate cartilage. Krejci, P., Krakow, D., Mekikian, P.B., Wilcox, W.R. Pediatr. Res. (2007) [Pubmed]
 
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