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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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H. Paul Ehrlich

Division of Plastic Surgery

Pennsylvania State University College of Medicine

Hershey

Pennsylvania

USA

[email]@psu.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Plastic Surgery, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, USA. 2000 - 2010
  • Irving S. Zubar Laboratory, Division of Plastic Surgery, Department of Surgery, Hershey Medical Center, USA. 2006
  • Department of Surgery, Milton S. Hershey Medical Center, Pennsylvania State University College of Medicine, Hershey 17033-0850, USA. 1999 - 2003

References

  1. Thymosin beta4 enhances repair by organizing connective tissue and preventing the appearance of myofibroblasts. Ehrlich, H.P., Hazard, S.W. Ann. N. Y. Acad. Sci. (2010) [Pubmed]
  2. The myofibroblast, cadherin, alpha smooth muscle actin and the collagen effect. Ehrlich, H.P., Allison, G.M., Leggett, M. Cell Biochem. Funct. (2006) [Pubmed]
  3. Gap junction communications influence upon fibroblast synthesis of Type I collagen and fibronectin. Ehrlich, H.P., Sun, B., Saggers, G.C., Kromath, F. J. Cell. Biochem. (2006) [Pubmed]
  4. Elucidating the mechanism of wound contraction: rapid versus sustained myosin ATPase activity in attached-delayed-released compared with free-floating fibroblast-populated collagen lattices. Paul Ehrlich, H., Sun, B., Kainth, K.S., Kromah, F. Wound. Repair. Regen (2006) [Pubmed]
  5. Dynamic changes appearing in collagen fibers during intrinsic tendon repair. Ehrlich, H.P., Lambert, P.A., Saggers, G.C., Myers, R.L., Hauck, R.M. Ann. Plast. Surg (2005) [Pubmed]
  6. Role for gap junctional intercellular communications in wound repair. Ehrlich, H.P., Diez, T. Wound. Repair. Regen (2003) [Pubmed]
  7. Topical platelet-derived growth factor in patients enhances wound closure in the absence of wound contraction. Ehrlich, H.P., Freedman, B.M. Cytokines Cell. Mol. Ther. (2002) [Pubmed]
  8. Vanadate ingestion increases the gain in wound breaking strength and leads to better organized collagen fibers in rats during healing. Ehrlich, H.P., Keefer, K.A., Maish, G.O., Myers, R.L., Mackay, D.R. Plast. Reconstr. Surg. (2001) [Pubmed]
  9. Increased gelsolin expression and retarded collagen lattice contraction with smooth muscle cells from Crohn's diseased intestine. Ehrlich, H.P., Allison, G.M., Page, M.J., Kolton, W.A., Graham, M. J. Cell. Physiol. (2000) [Pubmed]
  10. Cell coupling modulates the contraction of fibroblast-populated collagen lattices. Ehrlich, H.P., Gabbiani, G., Meda, P. J. Cell. Physiol. (2000) [Pubmed]
  11. Differences in the mechanism for high- versus moderate-density fibroblast-populated collagen lattice contraction. Ehrlich, H.P., Rittenberg, T. J. Cell. Physiol. (2000) [Pubmed]
  12. Vanadate and the absence of myofibroblasts in wound contraction. Ehrlich, H.P., Keefer, K.A., Myers, R.L., Passaniti, A. Arch. Surg (1999) [Pubmed]
 
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