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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Peter J. Mohler

Department of Pathology

Vanderbilt University Medical Center

Nashville

TN 37232

USA

[email]@vanderbilt.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pathology, Vanderbilt University Medical Center, Nashville, TN 37232, USA. 2005
  • Howard Hughes Medical Institute and Department of Cell Biology, Duke University Medical Center, Durham, NC 27710, USA. 2004

References

  1. Ankyrin-based cardiac arrhythmias: a new class of channelopathies due to loss of cellular targeting. Mohler, P.J., Bennett, V. Curr. Opin. Cardiol. (2005) [Pubmed]
  2. Defects in ankyrin-based cellular pathways in metazoan physiology. Mohler, P.J., Bennett, V. Front. Biosci. (2005) [Pubmed]
  3. Ankyrin-B coordinates the Na/K ATPase, Na/Ca exchanger, and InsP3 receptor in a cardiac T-tubule/SR microdomain. Mohler, P.J., Davis, J.Q., Bennett, V. PLoS Biol. (2005) [Pubmed]
  4. Dysfunction in ankyrin-based cellular pathways and human cardiac arrhythmia. Mohler, P.J., Lowe, J.S., Banks, S. Future. Cardiol (2005) [Pubmed]
  5. Nav1.5 E1053K mutation causing Brugada syndrome blocks binding to ankyrin-G and expression of Nav1.5 on the surface of cardiomyocytes. Mohler, P.J., Rivolta, I., Napolitano, C., LeMaillet, G., Lambert, S., Priori, S.G., Bennett, V. Proc. Natl. Acad. Sci. U.S.A. (2004) [Pubmed]
 
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