Petter Portin
Laboratory of Genetics
Department of Biology
University of Turku
20014 Turku
Finland
Name/email consistency: high
- Evidence based on studies of the mus309 mutant, deficient in DNA double-strand break repair, that meiotic crossing over in Drosophila melanogaster is a two-phase process. Portin, P. Genetica (2010)
- The effect of the mus309 mutation, defective in DNA double-strand break repair, on crossing over in Drosophila melanogaster suggests a mechanism for the centromere effect of crossing over. Portin, P. Genetica (2010)
- The effect of the mus309 mutation, defective in DNA double-strand break repair, on crossing over in Drosophila melanogaster suggests a mechanism for interference. Portin, P. Hereditas (2009)
- The elusive concept of the gene. Portin, P. Hereditas (2009)
- Evolution of man in the light of molecular genetics: a review. Part II. Regulation of gene function, evolution of speech and of brains. Portin, P. Hereditas (2008)
- Historical development of the concept of the gene. Portin, P. J. Med. Philos (2002)
- General outlines of the molecular genetics of the Notch signalling pathway in Drosophila melanogaster: a review. Portin, P. Hereditas (2002)
- Numb suppresses the negative complementation at the Notch locus of Drosophila melanogaster, suggesting a putative mechanism for negative complementation. Portin, P. Genet. Res. (2001)
- The occurrence of double strand DNA breaks is not the sole condition for meiotic crossing over in Drosophila melanogaster. Portin, P., Rantanen, M. Genetica (2000)
- Occurrence of double strand DNA breaks and pairing of homologous chromosomes are preconditions of crossing over in Drosophila melanogaster, and its timing. Portin, P., Rantanen, M. Genetica (2000)