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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Parminder J. S. Vig

Department of Neurology

University of Mississippi Medical Center


MS 39216



Name/email consistency: high



  • Department of Neurology, University of Mississippi Medical Center, Jackson, MS 39216, USA. 1999 - 2009


  1. Bergmann glial S100B activates myo-inositol monophosphatase 1 and Co-localizes to purkinje cell vacuoles in SCA1 transgenic mice. Vig, P.J., Shao, Q., Subramony, S.H., Lopez, M.E., Safaya, E. Cerebellum (2009) [Pubmed]
  2. Role of tissue transglutaminase type 2 in calbindin-D28k interaction with ataxin-1. Vig, P.J., Wei, J., Shao, Q., Hebert, M.D., Subramony, S.H., Sutton, L.T. Neurosci. Lett. (2007) [Pubmed]
  3. Intranasal administration of IGF-I improves behavior and Purkinje cell pathology in SCA1 mice. Vig, P.J., Subramony, S.H., D'Souza, D.R., Wei, J., Lopez, M.E. Brain Res. Bull. (2006) [Pubmed]
  4. Calcium homeostasis and spinocerebellar ataxia-1 (SCA-1). Vig, P.J., Subramony, S.H., McDaniel, D.O. Brain Res. Bull. (2001) [Pubmed]
  5. Relationship between ataxin-1 nuclear inclusions and Purkinje cell specific proteins in SCA-1 transgenic mice. Vig, P.J., Subramony, S.H., Qin, Z., McDaniel, D.O., Fratkin, J.D. J. Neurol. Sci. (2000) [Pubmed]
  6. The effects of calbindin D-28K and parvalbumin antisense oligonucleotides on the survival of cultured Purkinje cells. Vig, P.J., McDaniel, D.O., Subramony, S.H., Qin, Z. Res. Commun. Mol. Pathol. Pharmacol. (1999) [Pubmed]
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