Qubai Hu
Department of Pathology
University of Washington
Seattle 98195
USA
Name/email consistency: high
- Endoproteolytic cleavage of FE65 converts the adaptor protein to a potent suppressor of the sAPPalpha pathway in primates. Hu, Q., Wang, L., Yang, Z., Cool, B.H., Zitnik, G., Martin, G.M. J. Biol. Chem. (2005)
- A candidate molecular mechanism for the association of an intronic polymorphism of FE65 with resistance to very late onset dementia of the Alzheimer type. Hu, Q., Cool, B.H., Wang, B., Hearn, M.G., Martin, G.M. Hum. Mol. Genet. (2002)
- Broadly altered expression of the mRNA isoforms of FE65, a facilitator of beta amyloidogenesis, in Alzheimer cerebellum and other brain regions. Hu, Q., Jin, L.W., Starbuck, M.Y., Martin, G.M. J. Neurosci. Res. (2000)