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Pranela Rameshwar

UMDNJ-New Jersey Medical School

Dept of Medicine-Hematology/Oncology

MSB, E-579

185 South Orange Avenue

[email]@umdnj.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • UMDNJ-New Jersey Medical School, Dept of Medicine-Hematology/Oncology, MSB, E-579, 185 South Orange Avenue. 1997 - 2012
  • Department of Medicine, UMDNJ-New Jersey Medical School, Newark, NJ 07103, USA. 1997 - 2012
  • UMDNJ-New Jersey Medical School, MSB, 185 South Orange Ave, Newark, USA. 2007 - 2009
  • Department of Medicine-Hematology/Oncology, New Jersey Medical School, University of Medicine & Dentistry of New Jersey, Newark, USA. 2006

References

  1. The tachykinergic system as avenues for drug intervention. Rameshwar, P. Recent. Pat. CNS. Drug. Discov (2012) [Pubmed]
  2. Effects by anthrax toxins on hematopoiesis: a key role for cytokines as mediators. Rameshwar, P., Wong, E.W., Connell, N.D. Cytokine (2012) [Pubmed]
  3. Stem cells in cardiac repair in an inflammatory microenvironment. Rameshwar, P., Qiu, H., Vatner, S.F. Minerva. Cardioangiol (2010) [Pubmed]
  4. Casting doubt on the safety of "off-the-shelf" mesenchymal stem cells for cell therapy. Rameshwar, P. Mol. Ther. (2009) [Pubmed]
  5. Potential novel targets in breast cancer. Rameshwar, P. Curr. Pharm. Biotechnol (2009) [Pubmed]
  6. IFNgamma and B7-H1 in the immunology of mesenchymal stem cells. Rameshwar, P. Cell Res. (2008) [Pubmed]
  7. Implication of possible therapies targeted for the tachykinergic system with the biology of neurokinin receptors and emerging related proteins. Rameshwar, P. Recent. Pat. CNS. Drug. Discov (2007) [Pubmed]
  8. An in vitro method to study the effects of hematopoietic regulators during immune and blood cell development. Patel, N., Castillo, M., Rameshwar, P. Biol. Proced. Online (2007) [Pubmed]
  9. G protein-coupled receptors in haematopoietic disruption. Ramkissoon, S.H., Patel, H.J., Taborga, M., Rameshwar, P. Expert. Opin. Biol. Ther (2006) [Pubmed]
  10. Substance p-fibronectin-cytokine interactions in myeloproliferative disorders with bone marrow fibrosis. Rameshwar, P., Oh, H.S., Yook, C., Gascon, P., Chang, V.T. Acta Haematol. (2003) [Pubmed]
  11. Structural similarity between the bone marrow extracellular matrix protein and neurokinin 1 could be the limiting factor in the hematopoietic effects of substance P. Rameshwar, P., Gascon, P., Bandari, P.S., Joshi, D.D., Fernandes, A., Dang, A. Can. J. Physiol. Pharmacol. (2002) [Pubmed]
  12. Vasoactive intestinal peptide (VIP) inhibits the proliferation of bone marrow progenitors through the VPAC1 receptor. Rameshwar, P., Gascon, P., Oh, H.S., Denny, T.N., Zhu, G., Ganea, D. Exp. Hematol. (2002) [Pubmed]
  13. Mimicry between neurokinin-1 and fibronectin may explain the transport and stability of increased substance P immunoreactivity in patients with bone marrow fibrosis. Rameshwar, P., Joshi, D.D., Yadav, P., Qian, J., Gascon, P., Chang, V.T., Anjaria, D., Harrison, J.S., Song, X. Blood (2001) [Pubmed]
  14. Hematopoietic modulation by the tachykinins. Rameshwar, P., Gascón, P. Acta Haematol. (1997) [Pubmed]
  15. Substance P: a regulatory neuropeptide for hematopoiesis and immune functions. Rameshwar, P. Clin. Immunol. Immunopathol. (1997) [Pubmed]
  16. Hematopoietic regulation mediated by interactions among the neurokinins and cytokines. Rameshwar, P., Poddar, A., Gascón, P. Leuk. Lymphoma (1997) [Pubmed]
 
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