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Abdelhaq Rami

Dr Senckenbergische Anatomie

Anatomie III

Universitätsklinikum

Theodor-Stern-Kai 7

Germany

[email]@em.uni-frankfurt.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Institut für Zelluläre und Molekulare Anatomie (Anatomie III), Klinikum der Johann Wolfgang von Goethe-Universität, Theodor-Stern-Kai 7, 60590 Franfurt/Main, Germany. 2008 - 2012
  • Dr Senckenbergische Anatomie, Anatomie III, Universitätsklinikum, Theodor-Stern-Kai 7, Germany. 1998 - 2010
  • Institute of Anatomy III, Dr. Senckenbergische Anatomie, Faculty of Medicine, Clinic of the Johann-Wolfgang-Goethe-University, Germany. 2003 - 2008

References

  1. Alterations in the expression of the anti-apoptotic factor HAX-1 upon seizures-induced hippocampal injury in the neonatal rat brain. Rami, A., Kim, M., Niquet, J., Langhagen, A. Neurochem. Res. (2012) [Pubmed]
  2. Translocation of the serine protease Omi/HtrA2 from mitochondria into the cytosol upon seizure-induced hippocampal injury in the neonatal rat brain. Rami, A., Kim, M., Niquet, J. Neurochem. Res. (2010) [Pubmed]
  3. Review: autophagy in neurodegeneration: firefighter and/or incendiarist?. Rami, A. Neuropathol. Appl. Neurobiol. (2009) [Pubmed]
  4. Exploiting endogenous anti-apoptotic proteins for novel therapeutic strategies in cerebral ischemia. Rami, A., Bechmann, I., Stehle, J.H. Prog. Neurobiol. (2008) [Pubmed]
  5. Focal cerebral ischemia induces upregulation of Beclin 1 and autophagy-like cell death. Rami, A., Langhagen, A., Steiger, S. Neurobiol. Dis. (2008) [Pubmed]
  6. Upregulation of Beclin 1 in the ischemic penumbra. Rami, A. Autophagy (2008) [Pubmed]
  7. Apoptosis meets autophagy-like cell death in the ischemic penumbra: Two sides of the same coin?. Rami, A., Kögel, D. Autophagy (2008) [Pubmed]
  8. Synergetic effects of caspase 3 and mu-calpain in XIAP-breakdown upon focal cerebral ischemia. Rami, A., Agarwal, R., Spahn, A. Neurochem. Res. (2007) [Pubmed]
  9. Spatial resolution of phospholipid scramblase 1 (PLSCR1), caspase-3 activation and DNA-fragmentation in the human hippocampus after cerebral ischemia. Rami, A., Sims, J., Botez, G., Winckler, J. Neurochem. Int. (2003) [Pubmed]
  10. Post-ischemic activation of caspase-3 in the rat hippocampus: evidence of an axonal and dendritic localisation. Rami, A., Jansen, S., Giesser, I., Winckler, J. Neurochem. Int. (2003) [Pubmed]
  11. Ischemic neuronal death in the rat hippocampus: the calpain-calpastatin-caspase hypothesis. Rami, A. Neurobiol. Dis. (2003) [Pubmed]
  12. beta2-Adrenergic receptor responsiveness of the calpain-calpastatin system and attenuation of neuronal death in rat hippocampus after transient global ischemia. Rami, A., Volkmann, T., Agarwal, R., Schoninger, S., Nürnberger, F., Saido, T.C., Winckler, J. Neurosci. Res. (2003) [Pubmed]
  13. Effective reduction of neuronal death by inhibiting gap junctional intercellular communication in a rodent model of global transient cerebral ischemia. Rami, A., Volkmann, T., Winckler, J. Exp. Neurol. (2001) [Pubmed]
  14. mu-Calpain activation, DNA fragmentation, and synergistic effects of caspase and calpain inhibitors in protecting hippocampal neurons from ischemic damage. Rami, A., Agarwal, R., Botez, G., Winckler, J. Brain Res. (2000) [Pubmed]
  15. Muscarinic-receptor antagonist scopolamine rescues hippocampal neurons from death induced by glutamate. Rami, A., Krieglstein, J. Brain Res. (1998) [Pubmed]
 
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