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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Richard C. Bates

Division of Cancer Biology and Angiogenesis

Department of Pathology

Beth Israel Deaconess Medical Center and Harvard Medical School

Boston

USA

[email]@*.harvard.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Cancer Biology and Angiogenesis, Department of Pathology, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, USA. 2003 - 2005
  • Cancer Research Unit, Faculty of Medicine, The University of Newcastle, New South Wales 2308, Australia. 2001

References

  1. Transcriptional activation of integrin beta6 during the epithelial-mesenchymal transition defines a novel prognostic indicator of aggressive colon carcinoma. Bates, R.C., Bellovin, D.I., Brown, C., Maynard, E., Wu, B., Kawakatsu, H., Sheppard, D., Oettgen, P., Mercurio, A.M. J. Clin. Invest. (2005) [Pubmed]
  2. The epithelial-mesenchymal transition of colon carcinoma involves expression of IL-8 and CXCR-1-mediated chemotaxis. Bates, R.C., DeLeo, M.J., Mercurio, A.M. Exp. Cell Res. (2004) [Pubmed]
  3. Tumor necrosis factor-alpha stimulates the epithelial-to-mesenchymal transition of human colonic organoids. Bates, R.C., Mercurio, A.M. Mol. Biol. Cell (2003) [Pubmed]
  4. Flt-1-dependent survival characterizes the epithelial-mesenchymal transition of colonic organoids. Bates, R.C., Goldsmith, J.D., Bachelder, R.E., Brown, C., Shibuya, M., Oettgen, P., Mercurio, A.M. Curr. Biol. (2003) [Pubmed]
  5. A CD44 survival pathway triggers chemoresistance via lyn kinase and phosphoinositide 3-kinase/Akt in colon carcinoma cells. Bates, R.C., Edwards, N.S., Burns, G.F., Fisher, D.E. Cancer Res. (2001) [Pubmed]
 
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