Robert G. Bennett
Department of Internal Medicine
University of Nebraska Medical Center
Omaha
Nebr.
USA
Name/email consistency: high
- Relaxin and its role in the development and treatment of fibrosis. Bennett, R.G. Transl. Res (2009)
- Relaxin reduces fibrosis in models of progressive and established hepatic fibrosis. Bennett, R.G., Heimann, D.G., Tuma, D.J. Ann. N. Y. Acad. Sci. (2009)
- Degradation of relaxin family peptides by insulin-degrading enzyme. Bennett, R.G., Heimann, D.G., Hamel, F.G. Ann. N. Y. Acad. Sci. (2009)
- Relaxin receptors in hepatic stellate cells and cirrhotic liver. Bennett, R.G., Dalton, S.R., Mahan, K.J., Gentry-Nielsen, M.J., Hamel, F.G., Tuma, D.J. Biochem. Pharmacol. (2007)
- Relaxin receptor expression in hepatic stellate cells and in cirrhotic rat liver tissue. Bennett, R.G., Mahan, K.J., Gentry-Nielsen, M.J., Tuma, D.J. Ann. N. Y. Acad. Sci. (2005)
- Insulin inhibition of the proteasome is dependent on degradation of insulin by insulin-degrading enzyme. Bennett, R.G., Fawcett, J., Kruer, M.C., Duckworth, W.C., Hamel, F.G. J. Endocrinol. (2003)
- Inhibition of markers of hepatic stellate cell activation by the hormone relaxin. Bennett, R.G., Kharbanda, K.K., Tuma, D.J. Biochem. Pharmacol. (2003)
- Insulin inhibits the ubiquitin-dependent degrading activity of the 26S proteasome. Bennett, R.G., Hamel, F.G., Duckworth, W.C. Endocrinology (2000)
- Degradation of amylin by insulin-degrading enzyme. Bennett, R.G., Duckworth, W.C., Hamel, F.G. J. Biol. Chem. (2000)
