Ricardo Saban
Department of Physiology
The University of Oklahoma Health Sciences Center
Oklahoma City
OK 73104
USA
Name/email consistency: high
- VEGF signaling mediates bladder neuroplasticity and inflammation in response to BCG. Saban, M.R., Davis, C.A., Avelino, A., Cruz, F., Maier, J., Bjorling, D.E., Sferra, T.J., Hurst, R.E., Saban, R. BMC Physiol. (2011)
- Urothelial expression of neuropilins and VEGF receptors in control and interstitial cystitis patients. Saban, R., Saban, M.R., Maier, J., Fowler, B., Tengowski, M., Davis, C.A., Wu, X.R., Culkin, D.J., Hauser, P., Backer, J., Hurst, R.E. Am. J. Physiol. Renal Physiol. (2008)
- Mandatory role of proteinase-activated receptor 1 in experimental bladder inflammation. Saban, R., D'Andrea, M.R., Andrade-Gordon, P., Derian, C.K., Dozmorov, I., Ihnat, M.A., Hurst, R.E., Davis, C.A., Simpson, C., Saban, M.R. BMC Physiol. (2007)
- Regulatory network of inflammation downstream of proteinase-activated receptors. Saban, R., D'Andrea, M.R., Andrade-Gordon, P., Derian, C.K., Dozmorov, I., Ihnat, M.A., Hurst, R.E., Simpson, C., Saban, M.R. BMC Physiol. (2007)
- Bladder inflammatory transcriptome in response to tachykinins: neurokinin 1 receptor-dependent genes and transcription regulatory elements. Saban, R., Simpson, C., Vadigepalli, R., Memet, S., Dozmorov, I., Saban, M.R. BMC. Urol (2007)
- Transcription factor network downstream of protease activated receptors (PARs) modulating mouse bladder inflammation. Saban, R., Simpson, C., Davis, C.A., Dozmorov, I., Maier, J., Fowler, B., Ihnat, M.A., Hurst, R.E., Wershil, B.K., Saban, M.R. BMC Immunol. (2007)
- Visualization of lymphatic vessels through NF-kappaB activity. Saban, M.R., Mémet, S., Jackson, D.G., Ash, J., Roig, A.A., Israël, A., Saban, R. Blood (2004)
- LPS-sensory peptide communication in experimental cystitis. Saban, M.R., Saban, R., Hammond, T.G., Haak-Frendscho, M., Steinberg, H., Tengowski, M.W., Bjorling, D.E. Am. J. Physiol. Renal Physiol. (2002)
- Mast cells mediate substance P-induced bladder inflammation through an NK(1) receptor-independent mechanism. Saban, R., Gerard, N.P., Saban, M.R., Nguyen, N.B., DeBoer, D.J., Wershil, B.K. Am. J. Physiol. Renal Physiol. (2002)









