Disease relevance of Poison Control Centers

• The San Francisco Bay Area Regional Poison Control Center studied 189 cases of toxic workplace exposure occurring over a six-month period in the belief that Regional Poison Control Center reporting could be a useful surveillance measure for occupational disease [1].
• Review of the Duke Poison Control Center records for 1977 revealed an evening peak for preschool poisonings, while older victims had a late night maximum [2].
• To determine if there was any validity to the toxicity claims, 849,575 plant exposures reported to the American Association of Poison Control Centers were electronically analyzed [3].
• A review of the American Association of Poison Control Centers data for 1993 and 1994 identified five cases of hydrocarbon pneumonitis in which extracorporeal membrane oxygenation was used and two survived [4].
• METHODS: A prospective evaluation of all childhood (age 1-72 months) single ingestions of acetaminophen-containing products was performed by the Utah Poison Control Center. All patients ingesting 140 mg/ kg or greater or an unknown amount were referred for medical evaluation [5].

Psychiatry related information on Poison Control Centers

• These case reports are supplemented by data from the American Association of Poison Control Centers and the National Institute on Drug Abuse. Of 206 reported overdose exposures to trazodone, no deaths were recorded [6].

High impact information on Poison Control Centers

• METHOD: All mentions of desipramine, amitriptyline, imipramine, nortriptyline, and doxepin in children and adolescents recorded in the American Association of Poison Control Centers Toxic Exposure Surveillance System from 1983 to 2002 were analyzed [7].
• In addition, the Toxic Exposure Surveillance System (TESS) of the American Association of Poison Control Centers (AAPCC) database was searched for human exposure cases [8].
• According to the American Association of Poison Control Centers, the most commonly used antidotes are acetylcysteine, naloxone, atropine, deferoxamine (desferrioxamine) and antivenins [9].
• METHODS: Five-year prospective observational study of consecutive pediatric patients referred to a regional poison control center with a theophylline concentration (theo) greater than or equal to 30 micrograms/ml [10].
• Inquiries in hospitals and poison control centers did not reveal any cases of carbamate poisoning [11].

Chemical compound and disease context of Poison Control Centers

• We present a case of phenformin-induced lactic acidosis in which we were consulted at the local poison control center [12].
• A comparison was made among phenylpropanolamine, aspirin, acetaminophen and ibuprofen in terms of their relative safety, as measured by adverse reaction reports published since 1980, the five semiannual reports published by the Drug Abuse Warning Network during 1984-1986 and annual reports from Poison Control Centers from 1983-1986 [13].
• EDs and poison control centers nationwide will undoubtedly continue to manage GHB, GBL, and 1,4-BD toxicities [14].

Biological context of Poison Control Centers

• SETTING: The 1991 through 1993 National Electronic Injury Surveillance System data on emergency department visits compiled by the Consumer Product Safety Commission and the 1993 through 1995 Toxic Exposure Surveillance System data on calls to poison control centers compiled by the American Association of Poison Control Centers [15].
• The utility of Poison Control Center data for assessing toxic occupational exposures among young workers [16].
• BACKGROUND: The American Association of Poison Control Centers defines penetrance as the number of exposures per 1000 residents of a population during one year [17].
• Marked differences in cell volume, service area, and center resources continue to exist between poison centers certified by the American Association of Poison Control Centers as Regional Poison Centers and those not meeting these criteria [18].

Associations of Poison Control Centers with chemical compounds

• PARTICIPANTS: Hospitalized patients with acetaminophen exposure who were reported to the Long Island Poison Control Center between January 1993 and June 1994 [19].
• Characterization of methylphenidate exposures reported to a regional poison control center [20].
• STUDY OBJECTIVE: The purpose of this study was to provide the first description of the effects of ingestion of low-concentration hydrofluoric acid in a population reported to a regional poison control center [21].
• The regional poison control center recommended oral treatment with NAC 140 mg/kg, followed by maintenance doses of 70 mg/kg every 4 hours until the serum acetaminophen level was no longer detectable, rather than the standard 72-hour treatment regimen [22].
• Lithium poisoning from a poison control center perspective [23].

Gene context of Poison Control Centers

• The French Poison Control Centers' computer system. Part 1: Computer organization in French Poison Control Centers [24].

Analytical, diagnostic and therapeutic context of Poison Control Centers

• A retrospective study was conducted of zolpidem ingestion in dogs that were reported to the ASPCA Animal Poison Control Center (APCC) between January 1998 and July 2000 [25].

References