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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 

Dexamethasone-responsive hypertension in young women with suppressed renin and aldosterone.

Pronounced hypoaldosteronism was found in three young women with hypertension and symptoms of mineralocorticoid overproduction--i.e., hyporeninaemia, hypokalaemia, and a fall in blood-pressure after diuretic therapy. Plasma 11-deoxycorticosterone and 18-hydroxy-11-dooxycorticosterone concentrations were normal Treatment with dexamethasone induced a return to normal of blood-pressure and plasma-potassium and an increase in plasma-renin activity and urinary aldosterone excretion. The data suggest that hypertension in these patients is maintained by overproduction of an unknown adrenocorticotropindependent mineralocortocoid.[1]

References

  1. Dexamethasone-responsive hypertension in young women with suppressed renin and aldosterone. Hoefnagels, W.H., Drayer, J.I., Hofman, J.A., Kloppenborg, P.W., Smals, A.G., Benraad, T.J. Lancet (1978) [Pubmed]
 
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