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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Baerbel Rohrer

Department of Ophthalmology

Medical University of South Carolina

Charleston

SC 29425

United States

[email]@musc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Ophthalmology, Medical University of South Carolina, Charleston, SC 29425, United States. 2001 - 2011
  • Howard Hughes Medical Institute, University of California at San Francisco, San Francisco, California 94143, USA. 2001

References

  1. The alternative pathway is required, but not alone sufficient, for retinal pathology in mouse laser-induced choroidal neovascularization. Rohrer, B., Coughlin, B., Kunchithapautham, K., Long, Q., Tomlinson, S., Takahashi, K., Holers, V.M. Mol. Immunol. (2011) [Pubmed]
  2. A targeted inhibitor of the complement alternative pathway reduces RPE injury and angiogenesis in models of age-related macular degeneration. Rohrer, B., Long, Q., Coughlin, B., Renner, B., Huang, Y., Kunchithapautham, K., Ferreira, V.P., Pangburn, M.K., Gilkeson, G.S., Thurman, J.M., Tomlinson, S., Holers, V.M. Adv. Exp. Med. Biol. (2010) [Pubmed]
  3. A targeted inhibitor of the alternative complement pathway reduces angiogenesis in a mouse model of age-related macular degeneration. Rohrer, B., Long, Q., Coughlin, B., Wilson, R.B., Huang, Y., Qiao, F., Tang, P.H., Kunchithapautham, K., Gilkeson, G.S., Tomlinson, S. Invest. Ophthalmol. Vis. Sci. (2009) [Pubmed]
  4. Eliminating complement factor D reduces photoreceptor susceptibility to light-induced damage. Rohrer, B., Guo, Y., Kunchithapautham, K., Gilkeson, G.S. Invest. Ophthalmol. Vis. Sci. (2007) [Pubmed]
  5. Classical complement activation and acquired immune response pathways are not essential for retinal degeneration in the rd1 mouse. Rohrer, B., Demos, C., Frigg, R., Grimm, C. Exp. Eye Res. (2007) [Pubmed]
  6. Rod and cone pigment regeneration in RPE65-/- mice. Rohrer, B., Crouch, R. Adv. Exp. Med. Biol. (2006) [Pubmed]
  7. Cone opsin mislocalization in Rpe65-/- mice: a defect that can be corrected by 11-cis retinal. Rohrer, B., Lohr, H.R., Humphries, P., Redmond, T.M., Seeliger, M.W., Crouch, R.K. Invest. Ophthalmol. Vis. Sci. (2005) [Pubmed]
  8. Multidestructive pathways triggered in photoreceptor cell death of the rd mouse as determined through gene expression profiling. Rohrer, B., Pinto, F.R., Hulse, K.E., Lohr, H.R., Zhang, L., Almeida, J.S. J. Biol. Chem. (2004) [Pubmed]
  9. Functionally intact glutamate-mediated signaling in bipolar cells of the TRKB knockout mouse retina. Rohrer, B., Blanco, R., Marc, R.E., Lloyd, M.B., Bok, D., Schneeweis, D.M., Reichardt, L.F. Vis. Neurosci. (2004) [Pubmed]
  10. Correlation of regenerable opsin with rod ERG signal in Rpe65-/- mice during development and aging. Rohrer, B., Goletz, P., Znoiko, S., Ablonczy, Z., Ma, J.X., Redmond, T.M., Crouch, R.K. Invest. Ophthalmol. Vis. Sci. (2003) [Pubmed]
  11. Retarded outer segment development in TrkB knockout mouse retina organ culture. Rohrer, B., Ogilvie, J.M. Mol. Vis. (2003) [Pubmed]
  12. Does constitutive phosphorylation protect against photoreceptor degeneration in Rpe65-/- mice?. Rohrer, B., Ablonczy, Z., Znoiko, S., Redmond, M., Ma, J.X., Crouch, R. Adv. Exp. Med. Biol. (2003) [Pubmed]
  13. Neurotrophin receptor TrkB activation is not required for the postnatal survival of retinal ganglion cells in vivo. Rohrer, B., LaVail, M.M., Jones, K.R., Reichardt, L.F. Exp. Neurol. (2001) [Pubmed]
  14. Gene dosage effect of the TrkB receptor on rod physiology and biochemistry in juvenile mouse retina. Rohrer, B. Mol. Vis. (2001) [Pubmed]
 
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