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Jeffrey L. Salisbury

Department of Biochemistry and Molecular Biology

Tumor Biology Program

Mayo Clinic

Rochester

USA

[email]@mayo.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Biochemistry and Molecular Biology, Tumor Biology Program, Mayo Clinic, Rochester, USA. 2007
  • Tumor Biology Program, Mayo Clinic, Rochester, Minnesota. 1999 - 2004
  • Tumor Biology Program, Mayo Clinic College of Medicine, Mayo Clinic, Rochester, USA. 2004

References

  1. A mechanistic view on the evolutionary origin for centrin-based control of centriole duplication. Salisbury, J.L. J. Cell. Physiol. (2007) [Pubmed]
  2. Centrosomes: Sfi1p and centrin unravel a structural riddle. Salisbury, J.L. Curr. Biol. (2004) [Pubmed]
  3. Primary cilia: putting sensors together. Salisbury, J.L. Curr. Biol. (2004) [Pubmed]
  4. Centrosome amplification and the origin of chromosomal instability in breast cancer. Salisbury, J.L., D'Assoro, A.B., Lingle, W.L. J. Mammary. Gland. Biol. Neoplasia (2004) [Pubmed]
  5. Centrosomes: coiled-coils organize the cell center. Salisbury, J.L. Curr. Biol. (2003) [Pubmed]
  6. Centrosome size is controlled by centriolar SAS-4. Salisbury, J.L. Trends Cell Biol. (2003) [Pubmed]
  7. Centrin-2 is required for centriole duplication in mammalian cells. Salisbury, J.L., Suino, K.M., Busby, R., Springett, M. Curr. Biol. (2002) [Pubmed]
  8. The contribution of epigenetic changes to abnormal centrosomes and genomic instability in breast cancer. Salisbury, J.L. J. Mammary. Gland. Biol. Neoplasia (2001) [Pubmed]
  9. Microtubule nucleating capacity of centrosomes in tissue sections. Salisbury, J.L., Lingle, W.L., White, R.A., Cordes, L.E., Barrett, S. J. Histochem. Cytochem. (1999) [Pubmed]
 
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