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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Beatrice Louise Pool-Zobel

Department of Nutritional Toxicology

Institute for Nutrition

Friedrich Schiller University

Dornburger Strabetae 25

Germany

[email]@uni-jena.de

Name/email consistency: medium

 
 
 
 
 
 
 

Affiliations

  • Department of Nutritional Toxicology, Institute for Nutrition, Friedrich Schiller University, Dornburger Strabetae 25, Germany. 2000 - 2007
  • Institute of Nutritional Physiology, Federal Research Centre for Nutrition, Karlsruhe, Germany. 1998 - 1999
  • Friedrich-Schiller-University of Jena, Department of Nutritional Toxicology, Germany. 1999

References

  1. Overview of experimental data on reduction of colorectal cancer risk by inulin-type fructans. Pool-Zobel, B.L., Sauer, J. J. Nutr. (2007) [Pubmed]
  2. Butyrate may enhance toxicological defence in primary, adenoma and tumor human colon cells by favourably modulating expression of glutathione S-transferases genes, an approach in nutrigenomics. Pool-Zobel, B.L., Selvaraju, V., Sauer, J., Kautenburger, T., Kiefer, J., Richter, K.K., Soom, M., Wölfl, S. Carcinogenesis (2005) [Pubmed]
  3. Inulin-type fructans and reduction in colon cancer risk: review of experimental and human data. Pool-Zobel, B.L. Br. J. Nutr. (2005) [Pubmed]
  4. Modulation of xenobiotic metabolising enzymes by anticarcinogens -- focus on glutathione S-transferases and their role as targets of dietary chemoprevention in colorectal carcinogenesis. Pool-Zobel, B., Veeriah, S., Böhmer, F.D. Mutat. Res. (2005) [Pubmed]
  5. Genetic damage and repair in human rectal cells for biomonitoring: sex differences, effects of alcohol exposure, and susceptibilities in comparison to peripheral blood lymphocytes. Pool-Zobel, B.L., Dornacher, I., Lambertz, R., Knoll, M., Seitz, H.K. Mutat. Res. (2004) [Pubmed]
  6. Experimental evidences on the potential of prebiotic fructans to reduce the risk of colon cancer. Pool-Zobel, B., van Loo, J., Rowland, I., Roberfroid, M.B. Br. J. Nutr. (2002) [Pubmed]
  7. Isoflavonoids and lignans have different potentials to modulate oxidative genetic damage in human colon cells. Pool-Zobel, B.L., Adlercreutz, H., Glei, M., Liegibel, U.M., Sittlingon, J., Rowland, I., Wähälä, K., Rechkemmer, G. Carcinogenesis (2000) [Pubmed]
  8. Analysis of DNA strand breaks, oxidized bases, and glutathione S-transferase P1 in human colon cells from biopsies. Pool-Zobel, B.L., Abrahamse, S.L., Collins, A.R., Kark, W., Gugler, R., Oberreuther, D., Siegel, E.G., Treptow-van Lishaut, S., Rechkemmer, G. Cancer Epidemiol. Biomarkers Prev. (1999) [Pubmed]
  9. Anthocyanins are potent antioxidants in model systems but do not reduce endogenous oxidative DNA damage in human colon cells. Pool-Zobel, B.L., Bub, A., Schröder, N., Rechkemmer, G. Eur. J. Nutr (1999) [Pubmed]
  10. Mechanisms by which vegetable consumption reduces genetic damage in humans. Pool-Zobel, B.L., Bub, A., Liegibel, U.M., Treptow-van Lishaut, S., Rechkemmer, G. Cancer Epidemiol. Biomarkers Prev. (1998) [Pubmed]
 
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