Marcus Schuchmann
Department of Medicine
University of Mainz
Langenbeckstr. 1
55131 Mainz
Germany
Name/email consistency: high
- Histone deacetylase inhibition by valproic acid down-regulates c-FLIP/CASH and sensitizes hepatoma cells towards CD95- and TRAIL receptor-mediated apoptosis and chemotherapy. Schuchmann, M., Schulze-Bergkamen, H., Fleischer, B., Schattenberg, J.M., Siebler, J., Weinmann, A., Teufel, A., Wörns, M., Fischer, T., Strand, S., Lohse, A.W., Galle, P.R. Oncol. Rep. (2006)
- MORT1/FADD is involved in liver regeneration. Schuchmann, M., Ruckert, F., Garcia-Lazaro, J.F., Karg, A., Burg, J., Knorr, N., Siebler, J., Varfolomeev, E.E., Wallach, D., Schreiber, W., Lohse, A.W., Galle, P.R. World J. Gastroenterol. (2005)
- HSP60 and CpG-DNA-oligonucleotides differentially regulate LPS-tolerance of hepatic Kupffer cells. Schuchmann, M., Hermann, F., Herkel, J., van der Zee, R., Galle, P.R., Lohse, A.W. Immunol. Lett. (2004)
- Dominant negative MORT1/FADD rescues mice from CD95 and TNF-induced liver failure. Schuchmann, M., Varfolomeev, E.E., Hermann, F., Rueckert, F., Strand, D., Koehler, H., Strand, S., Lohse, A.W., Wallach, D., Galle, P.R. Hepatology (2003)