Susan M. Quirk
Department of Animal Science
Cornell University
434 Morrison Hall
Ithaca
USA
Name/email consistency: high
- Role of the cell cycle in regression of the corpus luteum. Quirk, S.M., Cowan, R.G., Harman, R.M. Reproduction (2013)
- The susceptibility of granulosa cells to apoptosis is influenced by oestradiol and the cell cycle. Quirk, S.M., Cowan, R.G., Harman, R.M. J. Endocrinol. (2006)
- Progesterone receptor and the cell cycle modulate apoptosis in granulosa cells. Quirk, S.M., Cowan, R.G., Harman, R.M. Endocrinology (2004)
- Ovarian follicular growth and atresia: the relationship between cell proliferation and survival. Quirk, S.M., Cowan, R.G., Harman, R.M., Hu, C.L., Porter, D.A. J. Anim. Sci. (2004)
- Responsiveness of mouse corpora luteal cells to Fas antigen (CD95)-mediated apoptosis. Quirk, S.M., Harman, R.M., Huber, S.C., Cowan, R.G. Biol. Reprod. (2000)
- Regulation of Fas antigen (Fas, CD95)-mediated apoptosis of bovine granulosa cells by serum and growth factors. Quirk, S.M., Harman, R.M., Cowan, R.G. Biol. Reprod. (2000)
- Potentiation of Fas-mediated apoptosis of murine granulosa cells by interferon-gamma, tumor necrosis factor-alpha, and cycloheximide. Quirk, S.M., Porter, D.A., Huber, S.C., Cowan, R.G. Endocrinology (1998)
- Fas antigen-mediated apoptosis of ovarian surface epithelial cells. Quirk, S.M., Cowan, R.G., Huber, S.H. Endocrinology (1997)