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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Anastasis Stephanou

Institute of Child Health

University College London

Medical Molecular Biology Unit




Name/email consistency: high



  • Institute of Child Health, University College London, Medical Molecular Biology Unit, London, UK. 2005
  • Medical Molecular Biology Unit, Institute of Child Health, University College London, 30 Guilford Street, United Kingdom. 2001 - 2004


  1. Opposing actions of STAT-1 and STAT-3. Stephanou, A., Latchman, D.S. Growth. Factors (2005) [Pubmed]
  2. Role of STAT-1 and STAT-3 in ischaemia/reperfusion injury. Stephanou, A. J. Cell. Mol. Med. (2004) [Pubmed]
  3. STAT-1: a novel regulator of apoptosis. Stephanou, A., Latchman, D.S. Int. J. Exp. Pathol (2003) [Pubmed]
  4. Antiapoptotic activity of the free caspase recruitment domain of procaspase-9: a novel endogenous rescue pathway in cell death. Stephanou, A., Scarabelli, T.M., Knight, R.A., Latchman, D.S. J. Biol. Chem. (2002) [Pubmed]
  5. The carboxyl-terminal activation domain of the STAT-1 transcription factor enhances ischemia/reperfusion-induced apoptosis in cardiac myocytes. Stephanou, A., Scarabelli, T.M., Townsend, P.A., Bell, R., Yellon, D., Knight, R.A., Latchman, D.S. FASEB J. (2002) [Pubmed]
  6. Activated STAT-1 pathway in the myocardium as a novel therapeutic target in ischaemia/reperfusion injury. Stephanou, A. Eur. Cytokine Netw. (2002) [Pubmed]
  7. Induction of apoptosis and Fas receptor/Fas ligand expression by ischemia/reperfusion in cardiac myocytes requires serine 727 of the STAT-1 transcription factor but not tyrosine 701. Stephanou, A., Scarabelli, T.M., Brar, B.K., Nakanishi, Y., Matsumura, M., Knight, R.A., Latchman, D.S. J. Biol. Chem. (2001) [Pubmed]
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