Yoshiyuki Minegishi
Department of Immune Regulation
Graduate School
Tokyo Medical and Dental University
Tokyo 113-8519
Japan
Name/email consistency: high
- Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome. Minegishi, Y., Saito, M., Nagasawa, M., Takada, H., Hara, T., Tsuchiya, S., Agematsu, K., Yamada, M., Kawamura, N., Ariga, T., Tsuge, I., Karasuyama, H. J. Exp. Med. (2009)
- Hyper-IgE syndrome. Minegishi, Y. Curr. Opin. Immunol. (2009)
- Defects in Jak-STAT-mediated cytokine signals cause hyper-IgE syndrome: lessons from a primary immunodeficiency. Minegishi, Y., Karasuyama, H. Int. Immunol. (2009)
- Genetic origins of hyper-IgE syndrome. Minegishi, Y., Karasuyama, H. Curr. Allergy. Asthma. Rep (2008)
- Hyperimmunoglobulin E syndrome and tyrosine kinase 2 deficiency. Minegishi, Y., Karasuyama, H. Curr. Opin. Allergy. Clin. Immunol (2007)
- Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome. Minegishi, Y., Saito, M., Tsuchiya, S., Tsuge, I., Takada, H., Hara, T., Kawamura, N., Ariga, T., Pasic, S., Stojkovic, O., Metin, A., Karasuyama, H. Nature (2007)
- Human tyrosine kinase 2 deficiency reveals its requisite roles in multiple cytokine signals involved in innate and acquired immunity. Minegishi, Y., Saito, M., Morio, T., Watanabe, K., Agematsu, K., Tsuchiya, S., Takada, H., Hara, T., Kawamura, N., Ariga, T., Kaneko, H., Kondo, N., Tsuge, I., Yachie, A., Sakiyama, Y., Iwata, T., Bessho, F., Ohishi, T., Joh, K., Imai, K., Kogawa, K., Shinohara, M., Fujieda, M., Wakiguchi, H., Pasic, S., Abinun, M., Ochs, H.D., Renner, E.D., Jansson, A., Belohradsky, B.H., Metin, A., Shimizu, N., Mizutani, S., Miyawaki, T., Nonoyama, S., Karasuyama, H. Immunity (2006)
