Shyng Shiou F. Yuan
Department of Medical Research and Department of Obstetrics and Gynecology
E-Da Hospital
Kaohsiung City 824
Taiwan
Name/email consistency: high
- Role of MRE11 in Cell Proliferation, Tumor Invasion, and DNA Repair in Breast Cancer. Yuan, S.S., Hou, M.F., Hsieh, Y.C., Huang, C.Y., Lee, Y.C., Chen, Y.J., Lo, S. J. Natl. Cancer Inst. (2012)
- High visfatin expression in breast cancer tissue is associated with poor survival. Lee, Y.C., Yang, Y.H., Su, J.H., Chang, H.L., Hou, M.F., Yuan, S.S. Cancer Epidemiol. Biomarkers Prev. (2011)
- Selective cytotoxicity of squamocin on T24 bladder cancer cells at the S-phase via a Bax-, Bad-, and caspase-3-related pathways. Yuan, S.S., Chang, H.L., Chen, H.W., Kuo, F.C., Liaw, C.C., Su, J.H., Wu, Y.C. Life Sci. (2006)
- Aberrant expression and possible involvement of the leptin receptor in endometrial cancer. Yuan, S.S., Tsai, K.B., Chung, Y.F., Chan, T.F., Yeh, Y.T., Tsai, L.Y., Su, J.H. Gynecol. Oncol. (2004)
- Ionizing radiation-induced Rad51 nuclear focus formation is cell cycle-regulated and defective in both ATM(-/-) and c-Abl(-/-) cells. Yuan, S.S., Chang, H.L., Lee, E.Y. Mutat. Res. (2003)
- Annonacin, a mono-tetrahydrofuran acetogenin, arrests cancer cells at the G1 phase and causes cytotoxicity in a Bax- and caspase-3-related pathway. Yuan, S.S., Chang, H.L., Chen, H.W., Yeh, Y.T., Kao, Y.H., Lin, K.H., Wu, Y.C., Su, J.H. Life Sci. (2003)
- Neocarzinostatin-induced Rad51 nuclear focus formation is cell cycle regulated and aberrant in AT cells. Yuan, S.S., Yang, Y.K., Chen, H.W., Chung, Y.F., Chang, H.L., Su, J.H. Toxicol. Appl. Pharmacol. (2003)
- Neocarzinostatin induces Mre11 phosphorylation and focus formation through an ATM- and NBS1-dependent mechanism. Yuan, S.S., Chang, H.L., Hou, M.F., Chan, T.F., Kao, Y.H., Wu, Y.C., Su, J.H. Toxicology (2002)
- Pax-2 interacts with RB and reverses its repression on the promoter of Rig-1, a Robo member. Yuan, S.S., Yeh, Y.T., Lee, E.Y. Biochem. Biophys. Res. Commun. (2002)
- Arsenic-induced Mre11 phosphorylation is cell cycle-dependent and defective in NBS cells. Yuan, S.S., Su, J.H., Hou, M.F., Yang, F.W., Zhao, S., Lee, E.Y. DNA Repair (Amst.) (2002)