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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Carolyn M. Klinge

Department of Biochemistry & Molecular Biology

Center for Genetics & Molecular Medicine

University of Louisville School of Medicine

Louisville

USA

[email]@louisville.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Biochemistry & Molecular Biology, Center for Genetics & Molecular Medicine, University of Louisville School of Medicine, Louisville, USA. 1999 - 2012

References

  1. Inhibition of non-small-cell lung cancer growth by combined fulvestrant and vandetanib. Klinge, C.M. Future. Oncol (2012) [Pubmed]
  2. Targeting the Intracellular MUC1 C-terminal Domain Inhibits Proliferation and Estrogen Receptor Transcriptional Activity in Lung Adenocarcinoma Cells. Klinge, C.M., Radde, B.N., Imbert-Fernandez, Y., Teng, Y., Ivanova, M.M., Abner, S.M., Martin, A.L. Mol. Cancer Ther. (2011) [Pubmed]
  3. Ligand-dependent differences in estrogen receptor beta-interacting proteins identified in lung adenocarcinoma cells corresponds to estrogenic responses. Ivanova, M., Abner, S., Pierce, W., Klinge, C. Proteome. Sci (2011) [Pubmed]
  4. Estrogen receptor alpha 46 is reduced in tamoxifen resistant breast cancer cells and re-expression inhibits cell proliferation and estrogen receptor alpha 66-regulated target gene transcription. Klinge, C.M., Riggs, K.A., Wickramasinghe, N.S., Emberts, C.G., McConda, D.B., Barry, P.N., Magnusen, J.E. Mol. Cell. Endocrinol. (2010) [Pubmed]
  5. Estrogenic control of mitochondrial function and biogenesis. Klinge, C.M. J. Cell. Biochem. (2008) [Pubmed]
  6. Resveratrol stimulates nitric oxide production by increasing estrogen receptor alpha-Src-caveolin-1 interaction and phosphorylation in human umbilical vein endothelial cells. Klinge, C.M., Wickramasinghe, N.S., Ivanova, M.M., Dougherty, S.M. FASEB J. (2008) [Pubmed]
  7. Resveratrol and estradiol rapidly activate MAPK signaling through estrogen receptors alpha and beta in endothelial cells. Klinge, C.M., Blankenship, K.A., Risinger, K.E., Bhatnagar, S., Noisin, E.L., Sumanasekera, W.K., Zhao, L., Brey, D.M., Keynton, R.S. J. Biol. Chem. (2005) [Pubmed]
  8. Estrogen response element-dependent regulation of transcriptional activation of estrogen receptors alpha and beta by coactivators and corepressors. Klinge, C.M., Jernigan, S.C., Mattingly, K.A., Risinger, K.E., Zhang, J. J. Mol. Endocrinol. (2004) [Pubmed]
  9. The agonist activity of tamoxifen is inhibited by the short heterodimer partner orphan nuclear receptor in human endometrial cancer cells. Klinge, C.M., Jernigan, S.C., Risinger, K.E. Endocrinology (2002) [Pubmed]
  10. Estrogen response element sequence impacts the conformation and transcriptional activity of estrogen receptor alpha. Klinge, C.M., Jernigan, S.C., Smith, S.L., Tyulmenkov, V.V., Kulakosky, P.C. Mol. Cell. Endocrinol. (2001) [Pubmed]
  11. Short heterodimer partner (SHP) orphan nuclear receptor inhibits the transcriptional activity of aryl hydrocarbon receptor (AHR)/AHR nuclear translocator (ARNT). Klinge, C.M., Jernigan, S.C., Risinger, K.E., Lee, J.E., Tyulmenkov, V.V., Falkner, K.C., Prough, R.A. Arch. Biochem. Biophys. (2001) [Pubmed]
  12. Estrogen receptor interaction with estrogen response elements. Klinge, C.M. Nucleic Acids Res. (2001) [Pubmed]
  13. The aryl hydrocarbon receptor interacts with estrogen receptor alpha and orphan receptors COUP-TFI and ERRalpha1. Klinge, C.M., Kaur, K., Swanson, H.I. Arch. Biochem. Biophys. (2000) [Pubmed]
  14. Estrogen receptor interaction with co-activators and co-repressors. Klinge, C.M. Steroids (2000) [Pubmed]
  15. Estrogen receptor binding to estrogen response elements slows ligand dissociation and synergistically activates reporter gene expression. Klinge, C.M. Mol. Cell. Endocrinol. (1999) [Pubmed]
  16. Role of estrogen receptor ligand and estrogen response element sequence on interaction with chicken ovalbumin upstream promoter transcription factor (COUP-TF). Klinge, C.M. J. Steroid Biochem. Mol. Biol. (1999) [Pubmed]
  17. The aryl hydrocarbon receptor (AHR)/AHR nuclear translocator (ARNT) heterodimer interacts with naturally occurring estrogen response elements. Klinge, C.M., Bowers, J.L., Kulakosky, P.C., Kamboj, K.K., Swanson, H.I. Mol. Cell. Endocrinol. (1999) [Pubmed]
 
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