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Franco Silvestris

DIMO, Department of Internal Medicine and Clinical Oncology

University of Bari

Italy

[email]@*.uniba.it

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • DIMO, Department of Internal Medicine and Clinical Oncology, University of Bari, Italy. 2000 - 2009
  • Department of Biomedical Sciences and Human Oncology, Section of Internal Medicine and Clinical Oncology, University of Bari, 70124 Bari, Italy. 2001 - 2007

References

  1. Bone-resorbing cells in multiple myeloma: osteoclasts, myeloma cell polykaryons, or both? Silvestris, F., Ciavarella, S., De Matteo, M., Tucci, M., Dammacco, F. Oncologist (2009) [Pubmed]
  2. Negative regulation of the osteoblast function in multiple myeloma through the repressor gene E4BP4 activated by malignant plasma cells. Silvestris, F., Cafforio, P., De Matteo, M., Calvani, N., Frassanito, M.A., Dammacco, F. Clin. Cancer Res. (2008) [Pubmed]
  3. Expression and function of the calcitonin receptor by myeloma cells in their osteoclast-like activity in vitro. Silvestris, F., Cafforio, P., De Matteo, M., Quatraro, C., Dammacco, F. Leuk. Res. (2008) [Pubmed]
  4. Myeloma bone disease: pathogenetic mechanisms and clinical assessment. Silvestris, F., Lombardi, L., De Matteo, M., Bruno, A., Dammacco, F. Leuk. Res. (2007) [Pubmed]
  5. Impaired osteoblastogenesis in myeloma bone disease: role of upregulated apoptosis by cytokines and malignant plasma cells. Silvestris, F., Cafforio, P., Calvani, N., Dammacco, F. Br. J. Haematol. (2004) [Pubmed]
  6. Enhancement of T cell apoptosis correlates with increased serum levels of soluble Fas (CD95/Apo-1) in active lupus. Silvestris, F., Grinello, D., Tucci, M., Cafforio, P., Dammacco, F. Lupus (2003) [Pubmed]
  7. Recent advances in understanding the pathogenesis of anemia in multiple myeloma. Silvestris, F., Tucci, M., Quatraro, C., Dammacco, F. Int. J. Hematol. (2003) [Pubmed]
  8. Negative regulation of erythroblast maturation by Fas-L(+)/TRAIL(+) highly malignant plasma cells: a major pathogenetic mechanism of anemia in multiple myeloma. Silvestris, F., Cafforio, P., Tucci, M., Dammacco, F. Blood (2002) [Pubmed]
  9. Serum elevations of soluble Fas (CD95/apo-I) concur in deregulating T cell apoptosis during active lupus disease. Silvestris, F., Williams, R.C., Calvani, N., Grinello, D., Tucci, M., Cafforio, P., Dammacco, F. Clin. Exp. Med. (2002) [Pubmed]
  10. Fas-L up-regulation by highly malignant myeloma plasma cells: role in the pathogenesis of anemia and disease progression. Silvestris, F., Tucci, M., Cafforio, P., Dammacco, F. Blood (2001) [Pubmed]
  11. Anti-Fas (CD95/Apo-I) autoantibodies and soluble Fas levels concur in T cell depletion in HIV type 1 infection. Silvestris, F., Grinello, D., Del Prete, A., Cafforio, P., Quarto, M., Dammacco, F. AIDS Res. Hum. Retroviruses (2001) [Pubmed]
  12. Immunogenicity of an eight amino acid domain shared by Fas (CD95/Apo-I) and HIV-1 gp120. I. Structural and antigenic analysis. Silvestris, F., Cocco, T., Cafforio, P., Calvani, N., Dammacco, F. Mol. Med. (2000) [Pubmed]
  13. VEINCTR-N, an immunogenic epitope of Fas (CD95/Apo-I), and soluble Fas enhance T-cell apoptosis in vitro. II. Functional analysis and possible implications in HIV-1 disease. Silvestris, F., Cafforio, P., Tucci, M., Del Prete, A., Dammacco, F. Mol. Med. (2000) [Pubmed]
 
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