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Antonio Ruiz-Vela

Howard Hughes Medical Institute

Department of Pathology and Medicine

Dana-Farber Cancer Institute

Harvard Medical School

USA

[email]@cnb.uam.es

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Howard Hughes Medical Institute, Department of Pathology and Medicine, Dana-Farber Cancer Institute, Harvard Medical School, USA. 2005
  • Department of Immunology Centro Nacional de Biotecnología, CSIC, Universidad Autónoma de Madrid, Campus de Cantoblanco, Spain. 1999 - 2002

References

  1. Proapoptotic BAX and BAK control multiple initiator caspases. Ruiz-Vela, A., Opferman, J.T., Cheng, E.H., Korsmeyer, S.J. EMBO Rep. (2005) [Pubmed]
  2. Nuclear Apaf-1 and cytochrome c redistribution following stress-induced apoptosis. Ruiz-Vela, A., González de Buitrago, G., Martínez-A, C. FEBS Lett. (2002) [Pubmed]
  3. Apaf-1 localization is modulated indirectly by Bcl-2 expression. Ruiz-Vela, A., Albar, J.P., Martínez, C.A. FEBS Lett. (2001) [Pubmed]
  4. Transplanted long-term cultured pre-BI cells expressing calpastatin are resistant to B cell receptor-induced apoptosis. Ruiz-Vela, A., Serrano, F., González, M.A., Abad, J.L., Bernad, A., Maki, M., Martínez-A, C. J. Exp. Med. (2001) [Pubmed]
  5. Implication of calpain in caspase activation during B cell clonal deletion. Ruiz-Vela, A., González de Buitrago, G., Martínez-A, C. EMBO J. (1999) [Pubmed]
 
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