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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Transactivation joins multiple tracks to the ERK/ MAPK cascade.

Many agonists of G-protein-coupled receptors (GPCRs) can stimulate receptor tyrosine kinases and the extracellular signal-regulated kinase (ERK)/mitogen- activated protein kinase ( MAPK) pathway. A 'transactivation' mechanism, which links these events in one signalling chain, inspired many researchers, but inevitably raised new questions. A 'multi-track' model for GPCR signalling to the ERK/ MAPK pathway might resolve some of the puzzles in the transactivation field.[1]

References

  1. Transactivation joins multiple tracks to the ERK/MAPK cascade. Wetzker, R., Böhmer, F.D. Nat. Rev. Mol. Cell Biol. (2003) [Pubmed]
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