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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Unexpected autocrine role of vascular endothelial growth factor in squamous cell carcinoma.

Mirones et al. demonstrate that keratinocytes deficient in VEGF are capable of forming tumors but use a distinct form of aneuploidy and signaling to form tumors. This knowledge is important because inhibitors of VEGF, including bevacizumab (anti-VEGF antibodies) and sorafenib (Braf/VEGFR2 kinase inhibitor), have already entered the clinic. These agents may "remodel" tumor signaling.[1]

References

  1. Unexpected autocrine role of vascular endothelial growth factor in squamous cell carcinoma. Arbiser, J.L. J. Invest. Dermatol. (2009) [Pubmed]
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