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Gene Review: 

ptsI  -  phosphoenolpyruvate-protein...

Salmonella enterica subsp. enterica serovar Typhimurium str. LT2

 
 
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High impact information on ptsI

  • These phenotypic properties are more extreme than those observed for tight ptsH and ptsI mutants, including mutants deleted for these genes [1].
  • Resistant ptsI mutants became sensitive to FOS when grown on D-glucose-6-phosphate, which induces an alternate transport system for FOS, or when grown in the presence of cyclic adenosine 3',5'-monophosphate [2].
  • Strains carrying "tight" ptsI point mutations and all mutants in which some or all of the ptsI gene was deleted were FOS resistant [2].

References

  1. Physiological consequences of the complete loss of phosphoryl-transfer proteins HPr and FPr of the phosphoenolpyruvate:sugar phosphotransferase system and analysis of fructose (fru) operon expression in Salmonella typhimurium. Feldheim, D.A., Chin, A.M., Nierva, C.T., Feucht, B.U., Cao, Y.W., Xu, Y.F., Sutrina, S.L., Saier, M.H. J. Bacteriol. (1990) [Pubmed]
  2. Fosfomycin resistance: selection method for internal and extended deletions of the phosphoenolpyruvate:sugar phosphotransferase genes of Salmonella typhimurium. Cordaro, J.C., Melton, T., Stratis, J.P., Atagün, M., Gladding, C., Hartman, P.E., Roseman, S. J. Bacteriol. (1976) [Pubmed]
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