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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 

Defective inactivation of leukotriene B4 in patients with Sjögren-Larsson syndrome.

In 6 patients with Sjögren-Larsson syndrome (SLS), the urinary excretion of leukotriene B(4) (LTB(4)) and omega-hydroxy-LTB(4) was found to be highly elevated, whereas omega-carboxy-LTB(4) was absent. This abnormal pattern of urinary excretion of LTB(4) and its metabolites appears to be unique to patients with SLS and offers a new approach to the diagnosis of this disorder. Moreover, defective inactivation of LTB(4) might be of pathophysiologic significance in the disease.[1]

References

  1. Defective inactivation of leukotriene B4 in patients with Sjögren-Larsson syndrome. Willemsen, M.A., de Jong, J.G., van Domburg, P.H., Rotteveel, J.J., Wanders, R.J., Mayatepek, E. J. Pediatr. (2000) [Pubmed]
 
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