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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Telomeres, p21 and the cancer-aging hypothesis.

Telomere dysfunction suppresses cancer through the p53 tumor suppressor pathway but also contributes to aging. A new study suggests that these effects of dysfunctional telomeres may be separable, such that aging-but not cancer suppression-depends on the p21 cell cycle inhibitor.[1]

References

  1. Telomeres, p21 and the cancer-aging hypothesis. Bell, J.F., Sharpless, N.E. Nat. Genet. (2007) [Pubmed]
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