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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Dr Chris Carter

[email]@yahoo.com

Name/email consistency: high

 
 
 
 
 

 

 

 

 

 

 

 

  • Alzheimer's disease genes and cholesterol [1]
  • Alzheimer's disease Genes and Herpes simplex [2]
  • APP, APOE, complement receptor 1, clusterin and PICALM and their involvement in the herpes simplex life cycle.  [3]
  • Alzheimer's disease plaques and tangles: Cemeteries of a Pyrrhic victory of the immune defence network against herpes simplex infection at the expense of complement and inflammation-mediated neuronal destruction.  [4]
  • Alzheimer's disease: a pathogenetic autoimmune disorder caused by herpes simplex in a gene-dependent manner [5].
  • Alzheimer's disease plaques and tangles: Cemeteries of a Pyrrhic victory of the immune defence network against herpes simplex infection at the expense of complement and inflammation-mediated neuronal destruction.  [4]
  • Schizophrenia Genes and glutamate [6]
  • Schizophrenia Genes and Herpes simplex [7]
  • Bipolar disorder genes and growth factor signalling [8]
  • EIF2B Pathogen and gene convergence in Bipolar disorder and Schizophrenia [9]
  • Epstein-Barr and other viral mimicry of autoantigens, myelin and vitamin D-related proteins and of EIF2B, the cause of vanishing white matter disease: massive mimicry of multiple sclerosis relevant proteins by the Synechococcus phage [10].
  • Pathogen and autoantigen homologous regions within the cystic fibrosis transmembrane conductance regulator (CFTR) protein suggest an autoimmune treatable component of cystic fibrosis [11] .
  • Vaccinia and other viruses with available vaccines show marked homology with the HIV-1 envelope glycoprotein: The prospect of using existing vaccines to stem the AIDS pandemic [12].
  • The Fox and the Rabbits—Environmental Variables and Population Genetics (1) Replication Problems in Association Studies and the Untapped Power of GWAS (2) Vitamin A Deficiency, Herpes Simplex Reactivation and Other Causes of Alzheimer's Disease  

 

 

 

 

 

 

References

  1. Convergence of genes implicated in Alzheimer's disease on the cerebral cholesterol shuttle: APP, cholesterol, lipoproteins, and atherosclerosis. Carter, C.J. Neurochem. Int. (2007) [Pubmed]
  2. Interactions between the products of the Herpes simplex genome and Alzheimer's disease susceptibility genes: relevance to pathological-signalling cascades. Carter, C.J. Neurochem. Int. (2008) [Pubmed]
  3. APP, APOE, complement receptor 1, clusterin and PICALM and their involvement in the herpes simplex life cycle. Carter, C.J. Neurosci. Lett. (2010) [Pubmed]
  4. Alzheimer's disease plaques and tangles: Cemeteries of a Pyrrhic victory of the immune defence network against herpes simplex infection at the expense of complement and inflammation-mediated neuronal destruction. Carter, C.J. Neurochem. Int. (2011) [Pubmed]
  5. Alzheimer's disease: a pathogenetic autoimmune disorder caused by herpes simplex in a gene-dependent manner. Carter, C.J. Int. J. Alzheimers. Dis. (2010) [Pubmed]
  6. Schizophrenia susceptibility genes converge on interlinked pathways related to glutamatergic transmission and long-term potentiation, oxidative stress and oligodendrocyte viability. Carter, C.J. Schizophr. Res. (2006) [Pubmed]
  7. Schizophrenia susceptibility genes directly implicated in the life cycles of pathogens: cytomegalovirus, influenza, herpes simplex, rubella, and Toxoplasma gondii. Carter, C.J. Schizophr. Bull. (2009) [Pubmed]
  8. Multiple genes and factors associated with bipolar disorder converge on growth factor and stress activated kinase pathways controlling translation initiation: implications for oligodendrocyte viability. Carter, C.J. Neurochem. Int. (2007) [Pubmed]
  9. eIF2B and oligodendrocyte survival: where nature and nurture meet in bipolar disorder and schizophrenia?. Carter, C.J. Schizophr. Bull. (2007) [Pubmed]
  10. Epstein-Barr and other viral mimicry of autoantigens, myelin and vitamin D-related proteins and of EIF2B, the cause of vanishing white matter disease: massive mimicry of multiple sclerosis relevant proteins by the Synechococcus phage. Carter, C.J. Immunopharmacol. Immunotoxicol. (2011) [Pubmed]
  11. Pathogen and autoantigen homologous regions within the cystic fibrosis transmembrane conductance regulator (CFTR) protein suggest an autoimmune treatable component of cystic fibrosis. Carter, C.J. FEMS. Immunol. Med. Microbiol. (2011) [Pubmed]
  12. Vaccinia and other viruses with available vaccines show marked homology with the HIV-1 envelope glycoprotein: The prospect of using existing vaccines to stem the AIDS pandemic. Carter, C.J. Immunopharmacol. Immunotoxicol. (2011) [Pubmed]
 
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