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MeSH Review:

Enterotoxemia

Desouza, I.A. et al., Sayeed, S. et al., Tengerdy, R.P., Dray, T.

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Disease relevance of Enterotoxemia

Vitamin E adjuvants provided greater immunoprotection against enterotoxemia and epididymitis in sheep than conventional vaccines [1].
Clostridium perfringens type A and beta2 toxin associated with enterotoxemia in a 5-week-old goat [2].

High impact information on Enterotoxemia

To model the rapid lethality of type D enterotoxemias, studies were conducted involving intravenous (i.v.) injection of genotype D vegetative supernatants into mice, which were then observed for neurotoxic distress [3].
CONCLUSIONS: These results demonstrate that SEA-induced mouse hind paw inflammation is a useful model for studying SEA-mediated enterotoxemia and may be sufficiently sensitive to differentiate between the effects of SEA and those of SE type B (SEB) [4].

References

The role of vitamin E in immune response and disease resistance. Tengerdy, R.P. Ann. N. Y. Acad. Sci. (1990) [Pubmed]
Clostridium perfringens type A and beta2 toxin associated with enterotoxemia in a 5-week-old goat. Dray, T. Can. Vet. J. (2004) [Pubmed]
Epsilon-toxin is required for most Clostridium perfringens type D vegetative culture supernatants to cause lethality in the mouse intravenous injection model. Sayeed, S., Fernandez-Miyakawa, M.E., Fisher, D.J., Adams, V., Poon, R., Rood, J.I., Uzal, F.A., McClane, B.A. Infect. Immun. (2005) [Pubmed]
The pharmacological profile of mouse hind paw inflammation induced by staphylococcal enterotoxin type A. Desouza, I.A., Ribeiro-DaSilva, G. Inflamm. Res. (1997) [Pubmed]

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