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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Molecular cloning and expression of rat torsinA in the normal and genetically dystonic (dt) rat.

Deletions within the TOR1A gene cause early-onset (DYT1) torsion dystonia. We have cloned and sequenced the rat cDNA homologue of TOR1A and found a 91% identity with the human sequence. Northern blot analysis detects a single transcript of approximately 1.5 kb. In situ hybridization reveals a widespread distribution of torsinA mRNA within brain. No mutations were identified in the coding region of the gene in the genetically dystonic (dt) rat.[1]

References

  1. Molecular cloning and expression of rat torsinA in the normal and genetically dystonic (dt) rat. Ziefer, P., Leung, J., Razzano, T., Shalish, C., LeDoux, M.S., Lorden, J.F., Ozelius, L., Breakefield, X.O., Standaert, D.G., Augood, S.J. Brain Res. Mol. Brain Res. (2002) [Pubmed]
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