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PCOLCE deletion and expression analyses in uterine leiomyomata.

Uterine leiomyomata (UL) are benign tumors affecting many women of reproductive age. Cytogenetic studies have indicated that a significant percentage of leiomyomata have chromosomal rearrangements, including those involving the long arm of chromosome 7. Several candidate genes that map to chromosome 7 have been studied for possible roles in the pathogenesis of these tumors. PCOLCE, a gene whose product is involved in the cleavage of type I procollagen C-propeptide, has been mapped to the critical interval on chromosome 7, band q22. Here we evaluate by reverse-transcriptase polymerase chain reaction (RT-PCR) and fluorescence in situ hybridization the expression and deletion status of PCOLCE in a series of karyotyped uterine leiomyomata.[1]

References

  1. PCOLCE deletion and expression analyses in uterine leiomyomata. Ligon, A.H., Scott, I.C., Takahara, K., Greenspan, D.S., Morton, C.C. Cancer Genet. Cytogenet. (2002) [Pubmed]
 
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