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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Arginase: marker, effector, or candidate gene for asthma?

Microarray analysis of the expression profiles of lung tissue in two murine models of asthma revealed high levels of arginase I and arginase II activity, in association with IL-4 and IL-13 overexpression, suggesting that arginine pathways are critical in the pathogenesis of asthma.[1]

References

  1. Arginase: marker, effector, or candidate gene for asthma? Vercelli, D. J. Clin. Invest. (2003) [Pubmed]
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